INT100768

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Context Info
Confidence 0.13
First Reported 2002
Last Reported 2002
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 1.37
Pain Relevance 0.40

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell adhesion (Itga1)
Anatomy Link Frequency
T cell 2
Itga1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Neuritis 4 94.28 High High
Sciatic nerve 2 86.40 High High
Disease Link Frequency Relevance Heat
Apoptosis 8 99.50 Very High Very High Very High
Adhesions 4 99.40 Very High Very High Very High
Experimental Autoimmune Neuritis 4 95.24 Very High Very High Very High
Disease 2 84.88 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The early increase of T cell apoptosis following disruption of VLA-4/VCAM-1 interaction may reflect a novel signaling component of proapoptotic pathways.
VLA-4 Binding (interaction) of in T cell associated with apoptosis
1) Confidence 0.13 Published 2002 Journal Acta Neuropathol. Section Abstract Doc Link 11810178 Disease Relevance 0.70 Pain Relevance 0.19
We characterized the early effects of anti-very late antigen (VLA-4) and its counterligand vascular cell adhesion molecule-1 (VCAM-1) antibody therapy on T cell infiltration and apoptosis in adoptive transfer experimental autoimmune neuritis of female Lewis rats.
VLA-4 Binding (counterligand) of in T cell associated with experimental autoimmune neuritis, apoptosis, neuritis and adhesions
2) Confidence 0.10 Published 2002 Journal Acta Neuropathol. Section Abstract Doc Link 11810178 Disease Relevance 0.67 Pain Relevance 0.20

General Comments

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