INT100820

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Context Info
Confidence 0.54
First Reported 2002
Last Reported 2002
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.85
Pain Relevance 0.19

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Fas) extracellular region (Fas) plasma membrane (Fas)
nucleus (Fas) cytoplasm (Fas)
Anatomy Link Frequency
plasma 2
Fas (Mus musculus)
Pain Link Frequency Relevance Heat
Bile 1 99.14 Very High Very High Very High
cytokine 1 82.32 Quite High
chemokine 1 81.12 Quite High
Paracetamol 1 56.96 Quite High
withdrawal 1 25.00 Low Low
Disease Link Frequency Relevance Heat
Apoptosis 3 99.92 Very High Very High Very High
Disease 1 98.56 Very High Very High Very High
Stress 2 96.16 Very High Very High Very High
Toxicity 2 94.40 High High
Hepatotoxicity 3 89.16 High High
Injury 2 87.20 High High
Targeted Disruption 1 42.08 Quite Low
Nash(non-alcoholic Steatohepatitis) 2 29.44 Quite Low
Hepatitis 1 25.36 Quite Low
Parkinson's Disease 1 25.00 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In cholestatic disease, endogenously generated bile acids produce hepatocellular apoptosis by stimulating Fas translocation from the cytoplasm to the plasma membrane where self-aggregation occurs to trigger apoptosis.
Positive_regulation (stimulating) of Localization (translocation) of Fas in plasma associated with bile, apoptosis and disease
1) Confidence 0.54 Published 2002 Journal Toxicol. Sci. Section Abstract Doc Link 11812920 Disease Relevance 0.85 Pain Relevance 0.19

General Comments

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