INT101009

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Context Info
Confidence 0.03
First Reported 2002
Last Reported 2005
Negated 1
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0.46
Pain Relevance 0.19

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (B4GALNT1) plasma membrane (B4GALNT1) carbohydrate metabolic process (B4GALNT1)
Anatomy Link Frequency
vessels 2
B4GALNT1 (Homo sapiens)
Pain Link Frequency Relevance Heat
tetrodotoxin 1 67.00 Quite High
cINOD 1 63.76 Quite High
Percutaneous transluminal coronary angioplasty 1 58.88 Quite High
guanethidine 1 57.48 Quite High
Disease Link Frequency Relevance Heat
Cardiovascular Disease 4 93.68 High High
Neointima 1 91.16 High High
Vasculitis 1 75.00 Quite High
INFLAMMATION 1 63.52 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The results suggest that phentolamine relaxes HCC by a nonadrenergic-noncholinergic mechanism dependent on nitric oxide synthase activity and activation of K(ATP)-channel.
Regulation (dependent) of Positive_regulation (activation) of synthase
1) Confidence 0.03 Published 2005 Journal Int. J. Impot. Res. Section Abstract Doc Link 15510188 Disease Relevance 0 Pain Relevance 0.10
Neither HCT-1026 nor flurbiprofen affected inducible NO synthase induction in injured vessels.
Neg (Neither) Regulation (affected) of Positive_regulation (induction) of NO synthase in vessels
2) Confidence 0.03 Published 2002 Journal Arterioscler. Thromb. Vasc. Biol. Section Abstract Doc Link 11834526 Disease Relevance 0.46 Pain Relevance 0.09

General Comments

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