INT101010

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Context Info
Confidence 0.06
First Reported 2002
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 5
Total Number 5
Disease Relevance 4.49
Pain Relevance 1.83

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (B4GALNT1) plasma membrane (B4GALNT1) carbohydrate metabolic process (B4GALNT1)
Anatomy Link Frequency
plaques 2
vessels 2
B4GALNT1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Angina 3 99.52 Very High Very High Very High
cINOD 9 98.80 Very High Very High Very High
Abeta 7 98.34 Very High Very High Very High
COX2 6 97.88 Very High Very High Very High
ischemia 3 97.76 Very High Very High Very High
Inflammation 33 97.60 Very High Very High Very High
Inflammatory mediators 2 97.12 Very High Very High Very High
Central nervous system 10 96.40 Very High Very High Very High
cytokine 3 94.40 High High
aspirin 1 80.64 Quite High
Disease Link Frequency Relevance Heat
Atherosclerotic Plaque 4 99.62 Very High Very High Very High
Cv General 3 Under Development 3 99.52 Very High Very High Very High
Diabetes Mellitus 8 98.24 Very High Very High Very High
Coronary Artery Disease 1 97.76 Very High Very High Very High
INFLAMMATION 40 97.60 Very High Very High Very High
Disease 29 96.90 Very High Very High Very High
Central Nervous System Disease 9 96.40 Very High Very High Very High
Alzheimer's Dementia 6 95.56 Very High Very High Very High
Necrosis 1 95.32 Very High Very High Very High
Cancer 1 94.96 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Moreover, accumulation of a marker of protein nitration nitrotyrosine, as well as the superoxide anion (O(2)(-)) levels and inducible nitric oxide synthase (iNOS), were evaluated.
Positive_regulation (accumulation) of Positive_regulation (inducible) of synthase
1) Confidence 0.06 Published 2004 Journal Diabetes Section Abstract Doc Link 15331549 Disease Relevance 1.26 Pain Relevance 0.36
Neither HCT-1026 nor flurbiprofen affected inducible NO synthase induction in injured vessels.
Positive_regulation (inducible) of Positive_regulation (induction) of NO synthase in vessels
2) Confidence 0.04 Published 2002 Journal Arterioscler. Thromb. Vasc. Biol. Section Abstract Doc Link 11834526 Disease Relevance 0.46 Pain Relevance 0.09
Nitrite was quantified as a measure for inducible nitric oxide synthase (iNOS) activity.
Positive_regulation (measure) of Positive_regulation (inducible) of synthase
3) Confidence 0.03 Published 2004 Journal J. Cardiovasc. Pharmacol. Section Abstract Doc Link 15076232 Disease Relevance 0.47 Pain Relevance 0.54
Recent studies show that amyloid beta (Abeta), a peptide extensively studied in Alzheimer's disease and that is cleaved from APP by beta- and gamma-secretase, and/or Abeta-like peptides are also present in human atherosclerotic plaques, in particular in activated, inducible nitric oxide synthase (iNOS) expressing perivascular macrophages that had phagocytized platelets.
Positive_regulation (activated) of Positive_regulation (inducible) of synthase in plaques associated with atherosclerotic plaque, alzheimer's dementia, disease and abeta
4) Confidence 0.03 Published 2006 Journal Cardiovascular & hematological disorders drug targets Section Abstract Doc Link 16724933 Disease Relevance 0.99 Pain Relevance 0.37
B) dependent pathways inducing expression of inducible NO synthase (iNOS) and of the inducible form of cyclooxygenase (COX2), among other inflammatory mediators [21].
Positive_regulation (inducing) of Positive_regulation (inducible) of NO synthase associated with inflammatory mediators and cox2
5) Confidence 0.01 Published 2010 Journal J Neuroinflammation Section Body Doc Link PMC2958904 Disease Relevance 1.32 Pain Relevance 0.47

General Comments

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