INT103279

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Context Info
Confidence 0.32
First Reported 2002
Last Reported 2002
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.31
Pain Relevance 0.27

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell-cell signaling (PGR, CCL5) cytoplasm (PGR, CCL5) nucleoplasm (PGR)
aging (CCL5) enzyme binding (PGR) DNA binding (PGR)
PGR (Homo sapiens)
CCL5 (Homo sapiens)
Pain Link Frequency Relevance Heat
endometriosis 2 78.24 Quite High
pain pelvic 2 76.64 Quite High
Inflammatory response 1 68.88 Quite High
Disease Link Frequency Relevance Heat
INFLAMMATION 2 80.88 Quite High
Endometriosis 3 77.96 Quite High
Reprotox - General 3 2 76.64 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In addition, its action via progesterone response element cis-elements, PR appeared to inhibit trans-activation of a nuclear factor-kappaB-responsive element, further suppressing RANTES expression.
PR Negative_regulation (suppressing) of Gene_expression (expression) of RANTES
1) Confidence 0.32 Published 2002 Journal J. Clin. Endocrinol. Metab. Section Abstract Doc Link 12050207 Disease Relevance 0.31 Pain Relevance 0.27

General Comments

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