INT107422

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Context Info
Confidence 0.37
First Reported 2002
Last Reported 2002
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.59

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Acot1) mitochondrion (Acot1) lipid metabolic process (Acot1)
cytoplasm (Acot1)
Anatomy Link Frequency
ACh neurons 2
Acot1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Dopamine 1 99.84 Very High Very High Very High
monoamine 2 96.88 Very High Very High Very High
cocaine 4 95.44 Very High Very High Very High
antagonist 5 95.28 Very High Very High Very High
fluoxetine 9 88.64 High High
agonist 1 25.00 Low Low
Serotonin 1 25.00 Low Low

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Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These data support the hypothesis that the enhanced serotonergic modulation of ACh neurons in pCOC-exposed animals is largely mediated by dopamine (DA) and reflect a major biochemical persistence of neurodevelopmental adaptations elicited by early cocaine exposure.
Positive_regulation (mediated) of Regulation (modulation) of ACh in ACh neurons associated with dopamine and cocaine
1) Confidence 0.37 Published 2002 Journal Prog. Neuropsychopharmacol. Biol. Psychiatry Section Abstract Doc Link 12502023 Disease Relevance 0 Pain Relevance 0.59

General Comments

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