INT110518

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Context Info
Confidence 0.26
First Reported 2003
Last Reported 2003
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 1.43
Pain Relevance 0.78

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Tnf, Prkce) mitochondrion (Prkce) extracellular space (Tnf)
nucleus (Prkce) cell division (Prkce) endoplasmic reticulum (Prkce)
Anatomy Link Frequency
sensory neurons 1
Tnf (Rattus norvegicus)
Prkce (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Inflammation 6 100.00 Very High Very High Very High
Hyperalgesia 8 99.80 Very High Very High Very High
intrathecal 2 81.40 Quite High
Kinase C 1 44.56 Quite Low
IPN 1 42.56 Quite Low
Disease Link Frequency Relevance Heat
Hyperalgesia 10 100.00 Very High Very High Very High
INFLAMMATION 3 100.00 Very High Very High Very High
Necrosis 3 83.28 Quite High
Cancer 3 82.92 Quite High
Inflammatory Pain 1 42.56 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Our results demonstrate that the generation of the primed state does not require production of hyperalgesia and that TNFalpha, which is generated during acute inflammation, can act on sensory neurons to induce hyperalgesic priming by activating neuronal PKC epsilon.
TNFalpha Positive_regulation (activating) of PKC epsilon in sensory neurons associated with hyperalgesia and inflammation
1) Confidence 0.26 Published 2003 Journal Eur. J. Neurosci. Section Abstract Doc Link 12752784 Disease Relevance 1.43 Pain Relevance 0.78

General Comments

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