INT110796

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Context Info
Confidence 0.49
First Reported 2003
Last Reported 2011
Negated 0
Speculated 1
Reported most in Body
Documents 1
Total Number 4
Disease Relevance 0.51
Pain Relevance 4.84

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Cacna1a) DNA binding (Cacna1a) transmembrane transport (Cacna1a)
cytoplasm (Cacna1a) cell death (Cacna1a) nucleus (Cacna1a)
Anatomy Link Frequency
adrenal gland 2
Cacna1a (Mus musculus)
Pain Link Frequency Relevance Heat
amygdala 378 100.00 Very High Very High Very High
long-term potentiation 222 100.00 Very High Very High Very High
qutenza 237 99.98 Very High Very High Very High
isoflurane 33 99.52 Very High Very High Very High
anesthesia 24 99.42 Very High Very High Very High
Pyramidal cell 18 94.48 High High
Hippocampus 18 90.24 High High
Kinase C 1 89.44 High High
Cannabinoid receptor 6 89.04 High High
antagonist 54 88.08 High High
Disease Link Frequency Relevance Heat
Diabetes Mellitus 8 100.00 Very High Very High Very High
Decapitation 3 71.44 Quite High
Targeted Disruption 7 57.92 Quite High
Nociception 6 44.72 Quite Low
Anxiety Disorder 21 27.20 Quite Low
Pain 15 5.00 Very Low Very Low Very Low
Depression 9 5.00 Very Low Very Low Very Low
Hyperalgesia 3 5.00 Very Low Very Low Very Low
Epilepsy 3 5.00 Very Low Very Low Very Low
Drug Induced Neurotoxicity 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Therefore, beta-endorphin release from the adrenal gland appears to be responsible for the lowering of plasma glucose in STZ-diabetic rats induced by caffeic acid, through the activation of alpha 1A -adrenoceptors.
Spec (appears) Positive_regulation (responsible) of Positive_regulation (activation) of alpha 1A in adrenal gland associated with diabetes mellitus
1) Confidence 0.49 Published 2003 Journal Horm. Metab. Res. Section Abstract Doc Link 12778369 Disease Relevance 0.44 Pain Relevance 0.17
After isoflurane anesthesia before euthanasia capsaicin caused a TRPV1-mediated increase in the magnitude of LA-LTP.
Positive_regulation (caused) of Positive_regulation (increase) of LA associated with anesthesia, qutenza, long-term potentiation, isoflurane and amygdala
2) Confidence 0.05 Published 2011 Journal PLoS ONE Section Abstract Doc Link PMC3020947 Disease Relevance 0 Pain Relevance 1.53
As shown in Fig. 7A, capsaicin caused a significant enhancement of the magnitude of EC-induced LA-LTP in the isoflurane group (control: 128.1±4.1% [n?
Positive_regulation (enhancement) of Positive_regulation (induced) of LA associated with qutenza, isoflurane, long-term potentiation and amygdala
3) Confidence 0.05 Published 2011 Journal PLoS ONE Section Body Doc Link PMC3020947 Disease Relevance 0.07 Pain Relevance 1.71
Since the mechanisms in induction of LA-LTP are dependent on the used afferents we have attempted to determine the capsaicin-induced effect on LA-LTP depending on the used input (EC stimulation versus intranuclear stimulation site (IN)) in horizontal slices.
Positive_regulation (dependent) of Positive_regulation (induction) of LA associated with qutenza, long-term potentiation and amygdala
4) Confidence 0.05 Published 2011 Journal PLoS ONE Section Body Doc Link PMC3020947 Disease Relevance 0 Pain Relevance 1.43

General Comments

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