INT111912

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Context Info
Confidence 0.04
First Reported 2003
Last Reported 2003
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.41
Pain Relevance 0.22

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (TAP1) mitochondrion (TAP1) endoplasmic reticulum (TAP1)
ATPase activity (TAP1) plasma membrane (TAP1) transmembrane transport (TAP1)
TAP1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Hsan 2 77.12 Quite High
Pain 1 68.92 Quite High
Disease Link Frequency Relevance Heat
Hereditary Sensory And Autonomic Neuropathies 2 77.12 Quite High
Pain 1 68.92 Quite High
Ataxia 1 68.00 Quite High
Neuropathic Pain 2 63.44 Quite High
Sprains And Strains 1 61.88 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Although we did not identify a mutation in the human CCT4 gene in a set of HSN patients, this result clearly demonstrates the pathological consequences of a defect in Cct4, a subunit of CCT (cytosolic chaperonin-containing t-complex peptide-1), involved in folding tubulin, actin and other cytosolic proteins.
Negative_regulation (defect) of peptide-1
1) Confidence 0.04 Published 2003 Journal Hum. Mol. Genet. Section Abstract Doc Link 12874111 Disease Relevance 0.41 Pain Relevance 0.22

General Comments

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