INT112968

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Context Info
Confidence 0.34
First Reported 2003
Last Reported 2010
Negated 0
Speculated 1
Reported most in Body
Documents 5
Total Number 6
Disease Relevance 7.79
Pain Relevance 1.81

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Gnb5) plasma membrane (Gnb5) nucleus (Gnb5)
signal transducer activity (Gnb5)
Anatomy Link Frequency
nerve 1
Gnb5 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 14 100.00 Very High Very High Very High
corticosteroid 4 99.76 Very High Very High Very High
Pain 135 95.68 Very High Very High Very High
sodium channel 2 83.16 Quite High
chemokine 8 75.00 Quite High
backache 6 71.48 Quite High
Visceral pain 1 57.44 Quite High
Neuritis 1 48.92 Quite Low
medulla 1 28.44 Quite Low
Demyelination 1 11.92 Low Low
Disease Link Frequency Relevance Heat
Syndrome 222 100.00 Very High Very High Very High
INFLAMMATION 13 100.00 Very High Very High Very High
Neuropathic Pain 27 99.92 Very High Very High Very High
Infection 53 99.52 Very High Very High Very High
Nervous System Injury 6 99.28 Very High Very High Very High
Chronic Inflammatory Demyelinating Polyradiculoneuropathy 10 98.04 Very High Very High Very High
Sprains And Strains 11 97.64 Very High Very High Very High
Pain 120 95.68 Very High Very High Very High
Disease 33 82.32 Quite High
Vomiting 1 77.84 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
A possible cause of a salutary effect of corticosteroids could be a reduction of the perineurial and endoneurial inflammatory reaction in GBS.
Negative_regulation (reduction) of GBS associated with corticosteroid, inflammation and syndrome
1) Confidence 0.34 Published 2007 Journal J Neurol Section Body Doc Link PMC2778673 Disease Relevance 2.35 Pain Relevance 1.61
There is circumstantial evidence that standard IVIG dose (2 g/kg bodyweight) is not, or not sufficiently, effective in some GBS patients because a proportion of GBS patients continues to deteriorate after a standard course of IVIG [13].
Negative_regulation (proportion) of GBS associated with syndrome
2) Confidence 0.06 Published 2010 Journal J Clin Immunol Section Body Doc Link PMC2883091 Disease Relevance 0.99 Pain Relevance 0
Host factors may influence this susceptibility to develop GBS or the extent of nerve damage and outcome.
Negative_regulation (develop) of GBS in nerve associated with nervous system injury and syndrome
3) Confidence 0.06 Published 2010 Journal J Clin Immunol Section Body Doc Link PMC2883091 Disease Relevance 1.20 Pain Relevance 0.04
We present a case of AMAN, a subtype of GBS, following infection with C. jejuni subspecies jejuni, biotype II O:19.
Negative_regulation (subtype) of GBS associated with syndrome, neuropathic pain and infection
4) Confidence 0.03 Published 2010 Journal J Med Case Reports Section Body Doc Link PMC2852392 Disease Relevance 1.88 Pain Relevance 0
In contrast, both binding and intracellular survival of GBS were not affected in Fc?
Negative_regulation (survival) of GBS
5) Confidence 0.02 Published 2010 Journal PLoS Pathogens Section Body Doc Link PMC2987830 Disease Relevance 0.08 Pain Relevance 0
These data implicate MCP-1 and IP-10 in the pathogenesis of GBS, and IP-10 and MIP-3beta in the pathogenesis of CIDP.
Spec (implicate) Negative_regulation (implicate) of GBS associated with chronic inflammatory demyelinating polyradiculoneuropathy and syndrome
6) Confidence 0.01 Published 2003 Journal J. Clin. Immunol. Section Abstract Doc Link 12959218 Disease Relevance 1.22 Pain Relevance 0.16

General Comments

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