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Context Info
Confidence 0.32
First Reported 2003
Last Reported 2003
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.87
Pain Relevance 0.37

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (PLAU, VTN) extracellular region (PLAU, VTN) peptidase activity (PLAU)
signal transduction (PLAU) cell adhesion (VTN) proteinaceous extracellular matrix (VTN)
Anatomy Link Frequency
PC-3 2
PLAU (Homo sapiens)
VTN (Homo sapiens)
Pain Link Frequency Relevance Heat
aspirin 9 99.98 Very High Very High Very High
cINOD 1 76.40 Quite High
Disease Link Frequency Relevance Heat
Adhesions 4 98.44 Very High Very High Very High
Prostate Cancer 4 96.44 Very High Very High Very High
Metastasis 3 83.84 Quite High
Pancreatic Cancer 1 80.00 Quite High
INFLAMMATION 1 76.16 Quite High
Cancer 3 73.92 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Altogether, our data suggests that aspirin inhibits the formation of uPA-uPAR-FN-alpha3beta1 and uPA-uPAR-VN-alphavbeta3 complexes, resulting in the suppression of cell adhesion and cell motility of the highly invasive prostate cancer cells PC-3.
Negative_regulation (inhibits) of uPA Binding (complexes) of VN in PC-3 associated with aspirin, prostate cancer and adhesions
1) Confidence 0.32 Published 2003 Journal Int. J. Oncol. Section Abstract Doc Link 14532966 Disease Relevance 0.87 Pain Relevance 0.37

General Comments

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