INT114472

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Context Info
Confidence 0.48
First Reported 2003
Last Reported 2009
Negated 3
Speculated 1
Reported most in Body
Documents 13
Total Number 16
Disease Relevance 7.95
Pain Relevance 3.20

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
blood 4
nerves 4
optic nerve 1
sympathetic nervous system 1
Izumo1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Acupuncture 646 100.00 Very High Very High Very High
Migraine 3 90.32 High High
Catecholamine 3 71.20 Quite High
Nerve growth factor 70 70.04 Quite High
ischemia 9 48.84 Quite Low
depression 3 41.60 Quite Low
anesthesia 13 25.00 Low Low
cva 6 5.00 Very Low Very Low Very Low
alcohol 3 5.00 Very Low Very Low Very Low
imagery 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Polycystic Ovary Syndrome 890 100.00 Very High Very High Very High
Glaucoma 78 99.46 Very High Very High Very High
Low Tension Glaucoma 111 99.28 Very High Very High Very High
Headache 3 90.32 High High
Coronary Heart Disease 10 75.76 Quite High
Ocular Hypertension 42 74.60 Quite High
Hypertension 36 72.16 Quite High
Anovulation 10 54.60 Quite High
Arteritic Anterior Ischaemic Optic Neuropathy 3 48.84 Quite Low
Disease 31 47.68 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
It is well known that ovarian sympathetic nerves take part in the regulation of OBF [20].
Regulation (regulation) of OBF in nerves
1) Confidence 0.48 Published 2004 Journal Reprod Biol Endocrinol Section Body Doc Link PMC411056 Disease Relevance 0.47 Pain Relevance 0.15
Low-frequency EA at 1.5 mA had no significant influence on either OBF or MAP in both the Control group and the PCO group compared to baseline (Fig. 1A and 1B).
Neg (no) Regulation (influence) of OBF associated with polycystic ovary syndrome and acupuncture
2) Confidence 0.48 Published 2004 Journal Reprod Biol Endocrinol Section Body Doc Link PMC411056 Disease Relevance 0.49 Pain Relevance 0.30
At 1.5 and 3 mA, there was no significant influence on either OBF or MAP in any of the study groups (Fig. 3A,3B,3C,3D).
Neg (no) Regulation (influence) of OBF
3) Confidence 0.35 Published 2004 Journal Reprod Biol Endocrinol Section Body Doc Link PMC411056 Disease Relevance 0.58 Pain Relevance 0.19
In addition, how changes in the systemic circulation affected OBF was evaluated by continuously recording blood pressure.
Regulation (affected) of OBF in blood
4) Confidence 0.24 Published 2003 Journal Auton Neurosci Section Abstract Doc Link 14614964 Disease Relevance 0 Pain Relevance 0.26
Both low- and high-frequency EA at 1.5 mA and high-frequency EA at 3 mA had no effect on OBF or mean arterial blood pressure (MAP).
Neg (no) Regulation (effect) of OBF in blood associated with acupuncture
5) Confidence 0.24 Published 2003 Journal Auton Neurosci Section Abstract Doc Link 14614964 Disease Relevance 0 Pain Relevance 0.41
Whether the ovarian sympathetic nerves were involved in OBF responses was elucidated by severance of the ovarian sympathetic nerves.
Regulation (responses) of OBF in nerves
6) Confidence 0.24 Published 2003 Journal Auton Neurosci Section Abstract Doc Link 14614964 Disease Relevance 0 Pain Relevance 0.22
These results further indicate that OBF in PCO rats is resistant to systemic circulatory changes.
Regulation (resistant) of OBF associated with polycystic ovary syndrome
7) Confidence 0.21 Published 2004 Journal Reprod Biol Endocrinol Section Body Doc Link PMC411056 Disease Relevance 1.04 Pain Relevance 0.13
The OBF response to low-frequency EA was less pronounced in the PCO rats, compared with the control rats.
Regulation (response) of OBF associated with polycystic ovary syndrome and acupuncture
8) Confidence 0.21 Published 2004 Journal Reprod Biol Endocrinol Section Body Doc Link PMC411056 Disease Relevance 0.31 Pain Relevance 0.29
The OBF responses to low-frequency EA stimulation were investigated after severance of the ovarian sympathetic nerves, the SON and OPN, to determine contribution of the ovarian sympathetic nerves to OBF responses in the Control group and the PCO group.
Spec (determine) Regulation (responses) of OBF in nerves associated with polycystic ovary syndrome and acupuncture
9) Confidence 0.21 Published 2004 Journal Reprod Biol Endocrinol Section Body Doc Link PMC411056 Disease Relevance 0.63 Pain Relevance 0.21
In this investigation, we aimed to study the effect of EA on OBF in order to elucidate the role of ovarian sympathetic nerves and the most optimal stimulation parameters in the rat model of PCO.
Regulation (effect) of OBF in nerves associated with polycystic ovary syndrome and acupuncture
10) Confidence 0.21 Published 2004 Journal Reprod Biol Endocrinol Section Body Doc Link PMC411056 Disease Relevance 0.56 Pain Relevance 0.31
The OBF responses in both Control and PCO rats were dependent on the frequency of the EA stimulation; that is, stimulation with a low frequency increased OBF, while stimulation with a high frequency decreased OBF in control rats.
Regulation (responses) of OBF associated with polycystic ovary syndrome and acupuncture
11) Confidence 0.21 Published 2004 Journal Reprod Biol Endocrinol Section Body Doc Link PMC411056 Disease Relevance 0.58 Pain Relevance 0.33
This study highlights the crucial role of the sympathetic nervous system on OBF responses in rats with EV induced PCO.


Regulation (responses) of OBF in sympathetic nervous system associated with polycystic ovary syndrome
12) Confidence 0.21 Published 2004 Journal Reprod Biol Endocrinol Section Body Doc Link PMC411056 Disease Relevance 0.32 Pain Relevance 0.08
How systemic circulatory changes affect OBF was evaluated by continuous recording of the blood pressure.
Regulation (affect) of OBF in blood
13) Confidence 0.21 Published 2004 Journal Reprod Biol Endocrinol Section Abstract Doc Link PMC411056 Disease Relevance 0.55 Pain Relevance 0.19
Various articles have shown that abnormal ocular blood flow (OBF) is a risk factor for glaucoma.1-5 It has also been suggested that a loss of autoregulation of OBF may be present in primary open angle glaucoma (POAG).6
Regulation (autoregulation) of OBF in blood associated with glaucoma
14) Confidence 0.00 Published 2009 Journal Korean Journal of Ophthalmology : KJO Section Body Doc Link PMC2655738 Disease Relevance 0.73 Pain Relevance 0.13
Previous studies have shown that glaucoma patients exhibited faulty autoregulation of OBF during posture change.35,36 Given this, we performed two separate analyses to determine which, if any, of the measured independent hemodynamic variables were related significantly to glaucomatous damage, in one case during the diurnal period and in the other case during the nocturnal period.
Regulation (autoregulation) of OBF associated with glaucoma
15) Confidence 0.00 Published 2009 Journal Korean Journal of Ophthalmology : KJO Section Body Doc Link PMC2655738 Disease Relevance 0.86 Pain Relevance 0
This link between low nocturnal perfusion pressure and glaucomatous damage might be compatible with the findings of previous studies indicating abnormal OBF autoregulation in patients with NTG, based on experiments testing short-term changes of OPP.35,36,38 During changes in posture, glaucoma patients exhibited an abnormal response in blood velocity in the central retinal artery.36 A significant association was also observed between optic nerve hemodynamic parameters and MAP in the glaucoma patients.
Regulation (autoregulation) of OBF in optic nerve associated with low tension glaucoma and glaucoma
16) Confidence 0.00 Published 2009 Journal Korean Journal of Ophthalmology : KJO Section Body Doc Link PMC2655738 Disease Relevance 0.83 Pain Relevance 0

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