INT114604

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Context Info
Confidence 0.41
First Reported 2003
Last Reported 2009
Negated 1
Speculated 2
Reported most in Body
Documents 2
Total Number 4
Disease Relevance 0
Pain Relevance 0.73

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Acvr2a)
Anatomy Link Frequency
neurons 1
astrocytes 1
Acvr2a (Rattus norvegicus)
Pain Link Frequency Relevance Heat
adenocard 9 99.86 Very High Very High Very High
Central nervous system 8 89.84 High High
GABAergic 2 75.96 Quite High
Enkephalin 4 75.12 Quite High
Action potential 6 70.84 Quite High
antagonist 39 42.64 Quite Low
agonist 22 18.92 Low Low
Nerve growth factor 28 5.00 Very Low Very Low Very Low
Inflammation 27 5.00 Very Low Very Low Very Low
cytokine 22 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 43 50.00 Quite Low
Stress 49 39.92 Quite Low
Retina Disease 11 31.52 Quite Low
Death 1 25.84 Quite Low
Apoptosis 3 23.76 Low Low
INFLAMMATION 28 5.00 Very Low Very Low Very Low
Cancer 22 5.00 Very Low Very Low Very Low
Pituitary Cancer 11 5.00 Very Low Very Low Very Low
Starvation 10 5.00 Very Low Very Low Very Low
Rheumatoid Arthritis 8 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Furthermore, expressions of A2A-R and the transgene were found in both neurons and astrocytes, suggesting that adenosine might mediate its function through A2A-R in both cell types.
Spec (might) Positive_regulation (mediate) of A2A-R in neurons associated with adenocard
1) Confidence 0.41 Published 2003 Journal Eur. J. Neurosci. Section Abstract Doc Link 14622213 Disease Relevance 0 Pain Relevance 0.35
Furthermore, expressions of A2A-R and the transgene were found in both neurons and astrocytes, suggesting that adenosine might mediate its function through A2A-R in both cell types.
Spec (might) Positive_regulation (mediate) of A2A-R in astrocytes associated with adenocard
2) Confidence 0.14 Published 2003 Journal Eur. J. Neurosci. Section Abstract Doc Link 14622213 Disease Relevance 0 Pain Relevance 0.35
This glycoprotein binds activin and in this way masks the binding domain of the activin molecule, preventing it from binding to the Act-R-II; consequently, activation of Act-R-I does not occur (472), resulting in bio-neutralisation of activin.
Positive_regulation (activation) of Act-R-I
3) Confidence 0.12 Published 2008 Journal Journal of Neuroendocrinology Section Body Doc Link PMC2229370 Disease Relevance 0 Pain Relevance 0
This was smaller than the latency of the VEP evoked in nondystrophic RCS rats without ChR2 gene induction.
Neg (without) Positive_regulation (induction) of ChR2
4) Confidence 0.04 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2772120 Disease Relevance 0 Pain Relevance 0.04

General Comments

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