INT114831

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Context Info
Confidence 0.65
First Reported 2001
Last Reported 2003
Negated 0
Speculated 0
Reported most in Abstract
Documents 3
Total Number 3
Disease Relevance 3.85
Pain Relevance 0.47

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (Pecam1) cell adhesion (Pecam1) plasma membrane (Pecam1)
Anatomy Link Frequency
endothelial cell 2
Pecam1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
ischemia 4 97.32 Very High Very High Very High
cva 5 90.24 High High
aspirin 7 83.76 Quite High
Percutaneous transluminal coronary angioplasty 27 78.44 Quite High
cytokine 13 75.00 Quite High
COX-2 inhibitor 2 25.00 Low Low
cINOD 2 25.00 Low Low
Angina 2 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Adhesions 31 100.00 Very High Very High Very High
Diabetes Mellitus 26 98.50 Very High Very High Very High
Shock 4 98.28 Very High Very High Very High
Myocardial Infarction 12 98.12 Very High Very High Very High
Coronary Artery Disease 5 97.76 Very High Very High Very High
Ulcers 26 90.92 High High
Cv General 3 Under Development 4 90.24 High High
Necrosis 3 82.08 Quite High
Cancer 3 81.72 Quite High
Peptic Ulcer 10 75.00 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The prolongation of the healing in diabetic animals was associated with an increase in gastric mucosal expression and release of TNFalpha, interleukin-1 beta (IL-1 beta), suppression of the vascular endothelial growth factor (VEGF), platelet endothelial cell adhesion molecule-1 (PECAM-1) and the mucosal overexpression of heat shock protein 70 (HSP 70).
Localization (release) of PECAM in endothelial cell associated with diabetes mellitus, shock and adhesions
1) Confidence 0.65 Published 2003 Journal Eur. J. Pharmacol. Section Abstract Doc Link 14642793 Disease Relevance 1.58 Pain Relevance 0.11
The prolongation of the healing in diabetic animals was associated with an increase in gastric mucosal expression and release of TNFalpha, interleukin-1 beta (IL-1 beta), suppression of the vascular endothelial growth factor (VEGF), platelet endothelial cell adhesion molecule-1 (PECAM-1) and the mucosal overexpression of heat shock protein 70 (HSP 70).
Localization (release) of platelet endothelial cell adhesion molecule-1 in endothelial cell associated with diabetes mellitus, shock and adhesions
2) Confidence 0.60 Published 2003 Journal Eur. J. Pharmacol. Section Abstract Doc Link 14642793 Disease Relevance 1.58 Pain Relevance 0.11
Both soluble ICAM-1 and PECAM-1 have been reported released into the circulation following reperfusion after acute myocardial ischemia, resulting in a transient increase within the first hours after reperfusion [28, 32].
Localization (released) of PECAM-1 associated with coronary artery disease and ischemia
3) Confidence 0.13 Published 2001 Journal Crit Care Section Body Doc Link PMC31578 Disease Relevance 0.68 Pain Relevance 0.25

General Comments

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