INT116730

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Context Info
Confidence 0.13
First Reported 2004
Last Reported 2008
Negated 0
Speculated 3
Reported most in Abstract
Documents 1
Total Number 3
Disease Relevance 1.32
Pain Relevance 1.19

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

intracellular (Rac1, Calca) cytoplasm (Rac1, Calca) aging (Calca)
enzyme binding (Rac1) GTPase activity (Rac1) cell proliferation (Rac1)
Anatomy Link Frequency
bladder 4
neurons 2
Rac1 (Rattus norvegicus)
Calca (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Botox 6 100.00 Very High Very High Very High
Calcitonin gene-related peptide 4 100.00 Very High Very High Very High
Neuropeptide 2 100.00 Very High Very High Very High
substance P 2 100.00 Very High Very High Very High
Inflammation 4 94.80 High High
interstitial cystitis 2 91.68 High High
neuropeptide release 2 75.00 Quite High
Migraine 1 75.00 Quite High
fifth nerve 1 75.00 Quite High
Disease Link Frequency Relevance Heat
Injury 4 98.48 Very High Very High Very High
Cystitis 2 95.76 Very High Very High Very High
Neurogenic Inflammation 2 95.24 Very High Very High Very High
Urinary Tract Disease 2 92.84 High High
Interstitial Cystitis 2 91.68 High High
INFLAMMATION 2 75.00 Quite High
Headache 1 75.00 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
OBJECTIVE: To determine the effect of botulinum toxin type A (BTX-A) on the release of the neuropeptides substance P (SP) and calcitonin gene-related peptide (CGRP) from isolated bladder preparations after acute injury with HCl and the induction of cyclophosphamide (CYP)-induced cystitis, as neurogenic inflammation has been increasingly identified in urological disorders such as interstitial cystitis.
botulinum toxin Spec (determine) Regulation (effect) of Localization (release) of CGRP in bladder associated with cystitis, calcitonin gene-related peptide, inflammation, neuropeptide, injury, urinary tract disease, interstitial cystitis, neurogenic inflammation, botox and substance p
1) Confidence 0.13 Published 2008 Journal BJU Int. Section Abstract Doc Link 18184328 Disease Relevance 0.62 Pain Relevance 0.45
OBJECTIVE: To determine the effect of botulinum toxin type A (BTX-A) on the release of the neuropeptides substance P (SP) and calcitonin gene-related peptide (CGRP) from isolated bladder preparations after acute injury with HCl and the induction of cyclophosphamide (CYP)-induced cystitis, as neurogenic inflammation has been increasingly identified in urological disorders such as interstitial cystitis.
botulinum toxin Spec (determine) Regulation (effect) of Localization (release) of calcitonin gene-related peptide in bladder associated with cystitis, calcitonin gene-related peptide, inflammation, neuropeptide, injury, urinary tract disease, interstitial cystitis, neurogenic inflammation, botox and substance p
2) Confidence 0.13 Published 2008 Journal BJU Int. Section Abstract Doc Link 18184328 Disease Relevance 0.62 Pain Relevance 0.45
OBJECTIVES: To determine the effect of botulinum toxin type A on calcitonin gene-related peptide secretion from cultured trigeminal ganglia neurons.
botulinum toxin Spec (determine) Regulation (effect) of Localization (secretion) of calcitonin in neurons associated with calcitonin gene-related peptide and botox
3) Confidence 0.01 Published 2004 Journal Headache Section Abstract Doc Link 14979881 Disease Relevance 0.07 Pain Relevance 0.29

General Comments

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