INT116801

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Context Info
Confidence 0.29
First Reported 2004
Last Reported 2004
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.10
Pain Relevance 1.02

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Camk2b) cytosol (Camk2b) cytoskeleton (Camk2b)
microtubule organizing center (Camk2b) cytoplasm (Camk2b)
Anatomy Link Frequency
spinal 2
Camk2b (Rattus norvegicus)
Pain Link Frequency Relevance Heat
long-term potentiation 16 100.00 Very High Very High Very High
spinal dorsal horn 7 89.44 High High
Spinal cord 1 86.92 High High
c fibre 4 75.00 Quite High
Sciatic nerve 1 73.76 Quite High
Hippocampus 1 40.44 Quite Low
nMDA receptor 2 31.36 Quite Low
Disease Link Frequency Relevance Heat
Anaerobic Bacterial Infections 1 99.98 Very High Very High Very High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We found that (1) the protein level of phospho-CaMKII increased at both 30 min and 3 h after LTP induction, while the total protein level of CaMK II increased at 3 h but not at 30 min after LTP induction. (2) Spinal application of KN-93 at 30 min prior to the tetanus blocked both LTP induction and the increase in phospho-CaMK II. (3) 30 min after LTP induction, spinal application of KN-93 depressed LTP and the level of phospho-CaMK II (n=3). (4) Spinal application of KN-93 at 3 h after LTP, however, affected neither the amplitude of the spinal LTP nor the level of phospho-CaMK II in the spinal dorsal horn.
Negative_regulation (blocked) of Positive_regulation (increase) of CaMK II in spinal associated with spinal dorsal horn, anaerobic bacterial infections and long-term potentiation
1) Confidence 0.29 Published 2004 Journal Sheng Li Xue Bao Section Abstract Doc Link 14985835 Disease Relevance 0.10 Pain Relevance 1.02

General Comments

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