INT117821

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Context Info
Confidence 0.34
First Reported 2004
Last Reported 2004
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 0.86
Pain Relevance 2.02

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Tacr1, Cd4) cell adhesion (Cd4) enzyme binding (Cd4)
response to stress (Tacr1) cytoplasm (Tacr1) signal transducer activity (Tacr1)
Anatomy Link Frequency
thymocyte 2
Tacr1 (Rattus norvegicus)
Cd4 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
antagonist 2 98.32 Very High Very High Very High
qutenza 26 98.18 Very High Very High Very High
substance P 28 97.54 Very High Very High Very High
Disease Link Frequency Relevance Heat
Death 4 94.92 High High
Apoptosis 6 94.68 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
SP administration reversed the CPS-mediated inhibitory effects on the total thymocyte number and subset distribution, namely CD4+ and CD4- CD8- cells, through its interaction with NK-1R as shown by concomitant NK-1R (SR140333) antagonist administration.
NK-1R Binding (interaction) of CD4 in thymocyte associated with qutenza, antagonist and substance p
1) Confidence 0.34 Published 2004 Journal Neuroimmunomodulation Section Abstract Doc Link 15067207 Disease Relevance 0.43 Pain Relevance 1.01
SP administration reversed the CPS-mediated inhibitory effects on the total thymocyte number and subset distribution, namely CD4+ and CD4- CD8- cells, through its interaction with NK-1R as shown by concomitant NK-1R (SR140333) antagonist administration.
NK-1R Binding (interaction) of CD4 in thymocyte associated with qutenza, antagonist and substance p
2) Confidence 0.34 Published 2004 Journal Neuroimmunomodulation Section Abstract Doc Link 15067207 Disease Relevance 0.43 Pain Relevance 1.01

General Comments

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