INT118372

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Context Info
Confidence 0.55
First Reported 2004
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 4
Total Number 4
Disease Relevance 3.31
Pain Relevance 0.15

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Fgf23) extracellular space (Fgf23) extracellular region (Fgf23)
Anatomy Link Frequency
intestine 1
Fgf23 (Mus musculus)
Pain Link Frequency Relevance Heat
Pain 6 97.60 Very High Very High Very High
Bile 16 14.00 Low Low
Somatostatin 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
Spinal cord 1 5.00 Very Low Very Low Very Low
agonist 1 5.00 Very Low Very Low Very Low
sodium channel 1 5.00 Very Low Very Low Very Low
Eae 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Benign Tumor 1 98.76 Very High Very High Very High
Rickets 176 98.64 Very High Very High Very High
Pain 6 97.60 Very High Very High Very High
Cancer 35 96.40 Very High Very High Very High
Hypophosphatemia 26 94.60 High High
Metabolic Disorder 8 94.56 High High
Targeted Disruption 37 91.92 High High
Disease 27 91.72 High High
Calcification 6 91.52 High High
Hypercalcemia 6 88.08 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
However, the mechanism regulating serum Fgf23 levels has been unclear.
Regulation (regulating) of Fgf23
1) Confidence 0.55 Published 2010 Journal Cell Tissue Res Section Body Doc Link PMC2948652 Disease Relevance 1.44 Pain Relevance 0
The rapid normalization of FGF-23 levels following removal of a benign tumour and the subsequent improvement in the biochemical and histological parameters of bone and mineral metabolism suggest that FGF-23 played a key role in this girl's disease.
Regulation (normalization) of FGF-23 associated with benign tumor and disease
2) Confidence 0.45 Published 2004 Journal Bone Section Abstract Doc Link 15121023 Disease Relevance 1.10 Pain Relevance 0.15
Thus, bone plays a central role in FGF23 control and plays an important part in the newly described kidney–intestine–bone hormonal axis controlling Pi homeostasis and bone mineralisation.
Regulation (control) of FGF23 in intestine
3) Confidence 0.27 Published 2008 Journal Eur J Pediatr Section Body Doc Link PMC2668657 Disease Relevance 0.25 Pain Relevance 0
However, the mechanisms of control of FGF23 and its role in normal Pi homoeostasis are unclear at present.
Regulation (control) of FGF23
4) Confidence 0.27 Published 2008 Journal Eur J Pediatr Section Body Doc Link PMC2668657 Disease Relevance 0.52 Pain Relevance 0

General Comments

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