INT118779

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Context Info
Confidence 0.47
First Reported 2004
Last Reported 2004
Negated 0
Speculated 1
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.05
Pain Relevance 1.20

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell proliferation (Pax3) nucleus (Pax3) DNA binding (Pax3)
Anatomy Link Frequency
spinal cord 2
Pax3 (Mus musculus)
Pain Link Frequency Relevance Heat
substance P 19 100.00 Very High Very High Very High
Antinociceptive 3 97.52 Very High Very High Very High
tail-flick 2 97.44 Very High Very High Very High
Spinal cord 5 96.04 Very High Very High Very High
intrathecal 1 94.96 High High
Pain 1 52.36 Quite High
Analgesic 2 25.00 Low Low
Disease Link Frequency Relevance Heat
Pain 1 52.36 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These results suggest that degradation of SP by spinal endopeptidase-24.11 may lead to the formation of SP(1-7), which has an ability to produce antinociceptive effects at the mouse spinal cord level.
Spec (may) Positive_regulation (lead) of Protein_catabolism (degradation) of SP in spinal cord associated with antinociceptive, spinal cord and substance p
1) Confidence 0.47 Published 2004 Journal Methods Find Exp Clin Pharmacol Section Abstract Doc Link 15148521 Disease Relevance 0.05 Pain Relevance 1.20

General Comments

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