INT119318

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Context Info
Confidence 0.02
First Reported 2004
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0.24
Pain Relevance 0.67

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

pigmentation (Atrn) extracellular space (Atrn) extracellular region (Atrn)
plasma membrane (Atrn) molecular_function (Atrn) cytoplasm (Atrn)
Anatomy Link Frequency
brain 2
Atrn (Rattus norvegicus)
Pain Link Frequency Relevance Heat
aspirin 9 99.98 Very High Very High Very High
Glutamate 2 99.84 Very High Very High Very High
sodium channel 2 99.24 Very High Very High Very High
Glutamate receptor 4 97.72 Very High Very High Very High
agonist 1 96.12 Very High Very High Very High
alcohol 1 91.16 High High
Analgesic 1 78.48 Quite High
cINOD 1 25.00 Low Low
Neuronal excitability 1 25.00 Low Low
Disease Link Frequency Relevance Heat
Convulsion 3 95.32 Very High Very High Very High
Body Weight 1 80.08 Quite High
Neuroblastoma 1 67.44 Quite High
INFLAMMATION 1 25.00 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Physical correlation of the membrane oxidative damage was evident as the Fourier Transformation Infra Red (FTIR) study of the Aspirin treated membrane, which include an increased proportion of gauche to trans conformer, shift in the methylene C-H asymmetric and symmetric stretching frequencies, C = O double bond stretching, NH bending, antisymmetric (N)-CH3 bending, C-N stretching and antisymmetric CNC stretching while there was no change in the CH2 wagging and twisting as well as in NH-bending amide bond I and II.
Positive_regulation (increased) of Positive_regulation (treated) of membrane associated with aspirin
1) Confidence 0.02 Published 2010 Journal Nutr Hosp Section Abstract Doc Link 20449540 Disease Relevance 0.08 Pain Relevance 0.28
Since the synaptic release of glutamate mainly depends upon the activation of membrane voltage-sensitive sodium channels (VSSCs), the nature of interactions between nefopam and these ionic channels was studied by evaluating the effects of nefopam on binding of 3H-batrachotoxinin, a specific ligand of the VSSCs in rat brain membrane preparations.
Positive_regulation (depends) of Positive_regulation (activation) of membrane in brain associated with glutamate and sodium channel
2) Confidence 0.01 Published 2004 Journal Brain Res. Section Abstract Doc Link 15193535 Disease Relevance 0.16 Pain Relevance 0.38

General Comments

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