INT119789

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.20
First Reported 2004
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 4
Total Number 6
Disease Relevance 0.32
Pain Relevance 1.44

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Chrna9)
Chrna9 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
agonist 15 100.00 Very High Very High Very High
antagonist 12 99.40 Very High Very High Very High
adenocard 15 98.88 Very High Very High Very High
Calcium channel 3 97.20 Very High Very High Very High
GABAergic 117 93.68 High High
tetrodotoxin 30 93.60 High High
imagery 15 5.00 Very Low Very Low Very Low
Hippocampus 9 5.00 Very Low Very Low Very Low
Neurotransmitter 6 5.00 Very Low Very Low Very Low
interneuron 6 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Vibrio Infection 3 90.24 High High
Ganglion Cysts 6 85.88 High High
Cognitive Disorder 9 77.88 Quite High
Repression 3 68.56 Quite High
Toxicity 3 46.04 Quite Low
Phototoxic Dermatitis 3 5.00 Very Low Very Low Very Low
Disseminated Intravascular Coagulation 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The results indicate that nAChR-induced [3H]-ACh release is triggered by the influx of Ca2+, independent of voltage-sensitive calcium channels, presumably directly through nAChRs located on myenteric axon terminals.
Localization (release) of nAChR-induced associated with calcium channel
1) Confidence 0.20 Published 2004 Journal Neurochem. Int. Section Abstract Doc Link 15234106 Disease Relevance 0 Pain Relevance 0.30
Pretreatment with adenosine deaminase (0.5 U/mL) or with the adenosine A(2A)-receptor antagonist, ZM 241385 (50 nM), enhanced nAChR-induced transmitter release.
Localization (release) of nAChR-induced associated with adenocard and antagonist
2) Confidence 0.20 Published 2004 Journal Neurochem. Int. Section Abstract Doc Link 15234106 Disease Relevance 0 Pain Relevance 0.35
The nAChR agonist, 1,1-dimethyl-4-phenylpiperazinium (DMPP, 1-30 microM, 3 min) increased [3H]-ACh release in a concentration-dependent manner.
Localization (release) of nAChR associated with agonist
3) Confidence 0.17 Published 2004 Journal Neurochem. Int. Section Abstract Doc Link 15234106 Disease Relevance 0 Pain Relevance 0.31
7-nAChR mobility
Localization (mobility) of nAChR
4) Confidence 0.05 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2901346 Disease Relevance 0.24 Pain Relevance 0.24
7-nAChR dependent on their subcellular localization.
Localization (localization) of nAChR
5) Confidence 0.05 Published 2010 Journal PLoS ONE Section Abstract Doc Link PMC2901346 Disease Relevance 0.08 Pain Relevance 0.16
7-nAChR clusters were colocalized with synapsin-1; 19.8±0.5% were apposed to synapsin-1 terminals, and 78.4±0.5% were independent of them; n?
Localization (colocalized) of nAChR
6) Confidence 0.05 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2901346 Disease Relevance 0 Pain Relevance 0.08

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox