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Context Info
Confidence 0.02
First Reported 2004
Last Reported 2004
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.33
Pain Relevance 0.26

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (Scgb1d2) molecular_function (Lnp) cellular_component (Lnp)
biological_process (Lnp)
Anatomy Link Frequency
central nervous system 1
Scgb1d2 (Rattus norvegicus)
Lnp (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Central nervous system 1 97.78 Very High Very High Very High
Clonidine 5 94.88 High High
Glutamate 1 90.00 High High
gABA 1 38.08 Quite Low
agonist 1 36.80 Quite Low
Disease Link Frequency Relevance Heat
Hypertension 2 87.04 High High
Increased Venous Pressure Under Development 3 86.64 High High
Hypotension 2 78.16 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The effects of the sympatho-inhibitory agents clonidine and rilmenidine, known to activate both alpha2-adrenoceptors and nonadrenergic I1-imidazoline binding sites (I1BS) in the central nervous system, were compared to those of dicyclopropylmethyl-(4,5-dimethyl-4,5-dihydro-3H-pyrrol-2-yl)-amine hydrochloride (LNP 509), which selectively binds to the I1BS.
I1BS Binding (binds) of LNP in central nervous system associated with central nervous system and clonidine
1) Confidence 0.02 Published 2004 Journal Braz. J. Med. Biol. Res. Section Abstract Doc Link 15448876 Disease Relevance 0.33 Pain Relevance 0.26

General Comments

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