INT122069

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Context Info
Confidence 0.02
First Reported 2004
Last Reported 2004
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.21
Pain Relevance 0.51

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Trpv1) transmembrane transport (Trpv1) lipid metabolic process (Trpv1)
cytosol (Trpv1) extracellular space (Gpx3) extracellular region (Gpx3)
Gpx3 (Mus musculus)
Trpv1 (Mus musculus)
Pain Link Frequency Relevance Heat
qutenza 5 96.96 Very High Very High Very High
Analgesic 1 96.32 Very High Very High Very High
nociceptor 2 74.00 Quite High
Inflammation 2 70.32 Quite High
Pain 1 25.00 Low Low
Disease Link Frequency Relevance Heat
Cv Unclassified Under Development 1 70.84 Quite High
INFLAMMATION 1 70.32 Quite High
Infection 1 69.76 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Extracellular Ca2+ -dependent desensitization of TRPV1 is thought to be one mechanism underlying the paradoxical effectiveness of capsaicin as an analgesic therapy.
Extracellular Negative_regulation (desensitization) of TRPV1 associated with qutenza and analgesic
1) Confidence 0.02 Published 2004 Journal Novartis Found. Symp. Section Abstract Doc Link 15469041 Disease Relevance 0.21 Pain Relevance 0.51

General Comments

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