INT122370

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Context Info
Confidence 0.30
First Reported 2004
Last Reported 2004
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.17
Pain Relevance 1.18

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

aging (Ngf) response to stress (Tacr1) cytoplasm (Tacr1)
signal transducer activity (Tacr1) extracellular space (Ngf) signal transduction (Ngf)
Anatomy Link Frequency
skin 2
Tacr1 (Rattus norvegicus)
Ngf (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Nerve growth factor 13 100.00 Very High Very High Very High
qutenza 7 99.98 Very High Very High Very High
agonist 3 96.96 Very High Very High Very High
substance P 1 94.96 High High
antagonist 2 89.24 High High
Hyperalgesia 1 54.92 Quite High
Inflammation 1 54.40 Quite High
cytokine 1 48.88 Quite Low
Disease Link Frequency Relevance Heat
Injury 1 56.12 Quite High
Hyperalgesia 1 54.92 Quite High
INFLAMMATION 1 54.40 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The results show that topical administration of capsaicin causes a primarily NK1 receptor-dependent increase in the NGF content of rat skin, which is susceptible to inhibition by beta adrenergic agonists.
Positive_regulation (causes) of NK1 receptor Positive_regulation (increase) of NGF in skin associated with nerve growth factor, qutenza and agonist
1) Confidence 0.30 Published 2004 Journal Pain Section Abstract Doc Link 15494187 Disease Relevance 0.17 Pain Relevance 1.18

General Comments

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