INT122787

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Context Info
Confidence 0.67
First Reported 2004
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 4
Total Number 4
Disease Relevance 0.78
Pain Relevance 2.81

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell proliferation (Nanog) nucleoplasm (Nanog) nucleolus (Nanog)
nucleus (Nanog) embryo development (Nanog) DNA binding (Nanog)
Anatomy Link Frequency
spinal cord 1
cortex 1
Nanog (Mus musculus)
Pain Link Frequency Relevance Heat
Enkephalin 27 100.00 Very High Very High Very High
Spinal cord 4 99.84 Very High Very High Very High
dorsal root ganglion 1 99.00 Very High Very High Very High
Ventral tegmentum 2 97.94 Very High Very High Very High
Nucleus accumbens 2 97.46 Very High Very High Very High
Nicotine 14 96.44 Very High Very High Very High
antinociception 5 95.00 High High
intrathecal 1 92.96 High High
narcan 2 91.00 High High
Endogenous opioid 1 72.92 Quite High
Disease Link Frequency Relevance Heat
Ganglion Cysts 1 98.52 Very High Very High Very High
Cancer 2 88.88 High High
Teratoma 4 85.04 High High
Anxiety Disorder 12 80.08 Quite High
Injury 14 24.64 Low Low
Myocardial Infarction 13 24.36 Low Low
Cicatrix 8 5.00 Very Low Very Low Very Low
Infarction 6 5.00 Very Low Very Low Very Low
Keloid Scars 2 5.00 Very Low Very Low Very Low
Stroke 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The preproenkephalin (ppENK) mRNA level in spinal cord, but not dorsal root ganglion, was significantly increased 2 h following nicotine administration and recovered to control level 4 h after nicotine (5 mg/kg, s.c.) administration.
Transcription (level) of ENK in spinal cord associated with ganglion cysts, dorsal root ganglion, nicotine and spinal cord
1) Confidence 0.67 Published 2008 Journal Brain Res. Section Abstract Doc Link 18048009 Disease Relevance 0.10 Pain Relevance 1.50
The nucleus accumbens, ventral tegmental area, basolateral (BLA), central (CEA) and medial amygdaloid nuclei, prelimbic and infralimbic cortex were assessed for fos-related antigen (FRA) and/or ENK mRNA as well as neuronal activation of ENK neurons (FRA/ENK).
Transcription (assessed) of ENK in cortex associated with ventral tegmentum, nucleus accumbens and enkephalin
2) Confidence 0.65 Published 2004 Journal Eur. J. Neurosci. Section Abstract Doc Link 15525282 Disease Relevance 0.27 Pain Relevance 0.77
In contrast, colonies of undifferentiated ESCs and high expression levels of Nanog and Pou5f1 were found in cultures of the sorted GFP(?)
Transcription (found) of Nanog
3) Confidence 0.51 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2902505 Disease Relevance 0.34 Pain Relevance 0
The present study assessed alterations in mesolimbic enkephalin (ENK) mRNA levels after predator [2,5-dihydro-2,4,5-trimethylethiazoline (TMT)] and non-predator (butyric acid) odor encounter and/or light-dark (LD) testing in CD-1 mice immediately, 24, 48 and 168 h after the initial odor encounter and/or LD testing.
Transcription (levels) of ENK associated with enkephalin
4) Confidence 0.51 Published 2004 Journal Eur. J. Neurosci. Section Abstract Doc Link 15525282 Disease Relevance 0.07 Pain Relevance 0.54

General Comments

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