INT125768

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Context Info
Confidence 0.48
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 0.71
Pain Relevance 0

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small molecule metabolic process (GLP1R, INS) extracellular space (INS) extracellular region (INS)
plasma membrane (GLP1R) carbohydrate metabolic process (INS) cell-cell signaling (INS)
GLP1R (Homo sapiens)
INS (Homo sapiens)
Pain Link Frequency Relevance Heat
agonist 4 25.00 Low Low
Paracetamol 2 25.00 Low Low
Disease Link Frequency Relevance Heat
Diabetes Mellitus 14 99.92 Very High Very High Very High
Hypoglycemia 2 25.00 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Preservation of the insulin response to parenteral glucagon-like peptide-1 (GLP-1), contrasting with lack of stimulation of insulin secretion by the other known incretin gastric inhibitory polypeptide (GIP), prompted studies with exogenous GLP-1 in recent-onset Type 1 diabetes.
Positive_regulation (Preservation) of GLP-1 Regulation (response) of insulin associated with diabetes mellitus
1) Confidence 0.48 Published 2005 Journal Regul. Pept. Section Abstract Doc Link 15780434 Disease Relevance 0.35 Pain Relevance 0
Preservation of the insulin response to parenteral glucagon-like peptide-1 (GLP-1), contrasting with lack of stimulation of insulin secretion by the other known incretin gastric inhibitory polypeptide (GIP), prompted studies with exogenous GLP-1 in recent-onset Type 1 diabetes.
Positive_regulation (Preservation) of glucagon-like peptide-1 Regulation (response) of insulin associated with diabetes mellitus
2) Confidence 0.42 Published 2005 Journal Regul. Pept. Section Abstract Doc Link 15780434 Disease Relevance 0.35 Pain Relevance 0

General Comments

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