INT127962

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.32
First Reported 2005
Last Reported 2008
Negated 0
Speculated 1
Reported most in Abstract
Documents 3
Total Number 3
Disease Relevance 2.67
Pain Relevance 3.69

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (GRHL3) DNA binding (GRHL3)
Anatomy Link Frequency
gall bladder 1
dorsal horn 1
GRHL3 (Homo sapiens)
Pain Link Frequency Relevance Heat
Somatostatin 16 100.00 Very High Very High Very High
Bile 15 100.00 Very High Very High Very High
dorsal root ganglion 6 100.00 Very High Very High Very High
spinal dorsal horn 4 100.00 Very High Very High Very High
electroacupuncture 14 99.86 Very High Very High Very High
Neuropathic pain 10 99.84 Very High Very High Very High
Eae 6 98.64 Very High Very High Very High
Sciatic nerve 2 95.04 Very High Very High Very High
Pain 25 93.08 High High
Analgesic 2 79.12 Quite High
Disease Link Frequency Relevance Heat
Ganglion Cysts 8 100.00 Very High Very High Very High
Neuropathic Pain 10 99.84 Very High Very High Very High
Injury 8 98.64 Very High Very High Very High
Gallbladder Disease 12 98.36 Very High Very High Very High
Pain 25 93.08 High High
Disease 3 65.68 Quite High
Muscular Spasm 4 41.28 Quite Low
Hypertension 1 34.28 Quite Low
Pancreatitis 7 34.00 Quite Low
Gallstones 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
However, EA could significantly enhance SOM expression in dorsal root ganglion (DRG) and spinal dorsal horn as well as ppSOM mRNA level in DRG of neuropathic pain rats.
Positive_regulation (enhance) of ppSOM in dorsal horn associated with ganglion cysts, dorsal root ganglion, spinal dorsal horn, neuropathic pain, somatostatin and electroacupuncture
1) Confidence 0.32 Published 2005 Journal Neurosci. Lett. Section Abstract Doc Link 15970377 Disease Relevance 0.93 Pain Relevance 1.71
The present data demonstrated that EA could activate endogenous SOM of neuropathic pain rats and this might be one of the mechanisms that underlie the effectiveness of EA in the treatment of neuropathic pain.
Positive_regulation (activate) of SOM associated with neuropathic pain, somatostatin and electroacupuncture
2) Confidence 0.26 Published 2005 Journal Neurosci. Lett. Section Abstract Doc Link 15970377 Disease Relevance 1.08 Pain Relevance 1.66
An abnormal gall bladder ejection fraction could be an indication for cholecystectomy, while a fatty meal-stimulated bile duct dilatation mandates SOM and possible sphincterotomy.[47] Of the patients who have documented SOD and an intact gall bladder and who are treated with sphincterotomy, first, only 43% have long-term pain relief; some additional patients eventually show response to cholecystectomy.[48]
Spec (possible) Positive_regulation (stimulated) of SOM in gall bladder associated with pain, bile and gallbladder disease
3) Confidence 0.22 Published 2008 Journal Saudi Journal of Gastroenterology : Official Journal of the Saudi Gastroenterology Association Section Body Doc Link PMC2702883 Disease Relevance 0.66 Pain Relevance 0.32

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox