INT12888

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Context Info
Confidence 0.55
First Reported 1991
Last Reported 1991
Negated 0
Speculated 1
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.21
Pain Relevance 0.94

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

peptidase activity (Cpe) extracellular space (Cpe) extracellular region (Cpe)
Golgi apparatus (Cpe)
Cpe (Mus musculus)
Pain Link Frequency Relevance Heat
Enkephalin 6 100.00 Very High Very High Very High
alcohol 3 98.88 Very High Very High Very High
MU agonist 1 79.32 Quite High
Pain threshold 1 76.68 Quite High
Endogenous opioid 2 75.00 Quite High
Disease Link Frequency Relevance Heat
Pain 1 76.68 Quite High
Sprains And Strains 1 74.44 Quite High
Genetic Predisposition To Disease 1 63.04 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These results suggest that a relative lack of enkephalin peptides trans-synaptically, possibly resulting from enhanced enkephalin degradation may contribute to increase alcohol consumption in C57BL/6J mice.
Spec (possibly) Positive_regulation (enhanced) of Protein_catabolism (degradation) of enkephalin associated with enkephalin and alcohol
1) Confidence 0.55 Published 1991 Journal Alcohol. Clin. Exp. Res. Section Abstract Doc Link 1656811 Disease Relevance 0.21 Pain Relevance 0.94

General Comments

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