INT130469

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Context Info
Confidence 0.05
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.44
Pain Relevance 0.50

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

peptidase activity (Naalad2) cellular_component (Naalad2)
Tubgcp2 (Mus musculus)
Naalad2 (Mus musculus)
Pain Link Frequency Relevance Heat
Glutamate 7 100.00 Very High Very High Very High
Pain threshold 1 76.00 Quite High
peripheral neuropathy 1 75.00 Quite High
Sciatic nerve 2 60.00 Quite High
Disease Link Frequency Relevance Heat
Injury 3 89.32 High High
Pain 1 76.00 Quite High
Peripheral Neuropathy 2 75.00 Quite High
Brain Hemorrhage 1 75.00 Quite High
Cognitive Disorder 1 74.72 Quite High
Middle Cerebral Artery Infarction 1 34.24 Quite Low
Stroke 2 30.64 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
GCP II is believed to mediate the hydrolysis of N-acetyl-aspartyl-glutamate (NAAG) to glutamate and N-acetyl-aspartate, and inhibition of NAAG peptidase activity (by GCP II and other peptidases) is neuroprotective.
GCP II Negative_regulation (inhibition) of NAAG associated with glutamate
1) Confidence 0.05 Published 2005 Journal J. Neurochem. Section Abstract Doc Link 16190866 Disease Relevance 0.44 Pain Relevance 0.50

General Comments

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