INT131819

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Context Info
Confidence 0.15
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.44
Pain Relevance 0.46

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Cpox, Nfkb1) cytoplasm (Cpox, Nfkb1) DNA binding (Nfkb1)
protein complex (Nfkb1) cytosol (Nfkb1) signal transduction (Nfkb1)
Cpox (Mus musculus)
Nfkb1 (Mus musculus)
Pain Link Frequency Relevance Heat
Hyperalgesia 4 99.90 Very High Very High Very High
Inflammation 6 99.64 Very High Very High Very High
COX-2 inhibitor 4 73.28 Quite High
Analgesic 1 25.00 Low Low
Disease Link Frequency Relevance Heat
Hyperalgesia 4 99.90 Very High Very High Very High
INFLAMMATION 3 99.64 Very High Very High Very High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We examined whether nitric oxide (NO), derived from constitutive NO synthase (NOS) and/or inducible NOS (iNOS), could contribute to endotoxin-induced inflammatory hyperalgesia via interacting with nuclear factor-kappaB (NF-kappaB), inducible cyclooxygenase (COX-2) and/or polyADP-ribose synthase (PARS).
COX Binding (interacting) of NF-kappaB associated with hyperalgesia and inflammation
1) Confidence 0.15 Published 2006 Journal Pharmacol. Res. Section Abstract Doc Link 16310374 Disease Relevance 0.44 Pain Relevance 0.46

General Comments

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