INT135033

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Context Info
Confidence 0.65
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.16
Pain Relevance 1.51

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Hk1) mitochondrion (Hk1) nucleus (Hk1)
carbohydrate metabolic process (Hk1) protein complex (Hk1) kinase activity (Hk1)
Hk1 (Mus musculus)
Pain Link Frequency Relevance Heat
Endogenous opioid 2 99.96 Very High Very High Very High
analgesia 1 99.32 Very High Very High Very High
Opioid 4 99.12 Very High Very High Very High
Pain 2 92.40 High High
Analgesic 2 91.56 High High
narcan 3 89.84 High High
antagonist 1 89.48 High High
opioid receptor 1 89.04 High High
Antinociceptive 4 86.00 High High
Morphine 3 84.32 Quite High
Disease Link Frequency Relevance Heat
Pain 2 92.40 High High
Nociception 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Consistent with previous biochemical data, a likely mechanism underlying the peptide-mediated enhancement of opioid analgesia may center on the ability of r/m HK-1 to release endogenous opioid peptides.
Localization (release) of HK-1 associated with endogenous opioid, opioid and analgesia
1) Confidence 0.65 Published 2006 Journal Behav. Brain Res. Section Abstract Doc Link 16621052 Disease Relevance 0.16 Pain Relevance 1.51

General Comments

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