INT135566

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Context Info
Confidence 0.01
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.45
Pain Relevance 0.18

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Atp2b4)
Anatomy Link Frequency
myocyte 2
Atp2b4 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
ischemia 3 99.40 Very High Very High Very High
tetrodotoxin 1 60.00 Quite High
Disease Link Frequency Relevance Heat
Cv Unclassified Under Development 3 99.40 Very High Very High Very High
Injury 2 94.88 High High
Hypoxia 1 89.12 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
During ischemia, myocyte Ca(2+) overload can contribute to ATP depletion by activation of Ca(2+)-dependent ATPases and by induction of the mitochondrial permeability transition, and thus is an important contributor to myocyte dysfunction and injury.
Positive_regulation (contribute) of Positive_regulation (activation) of ATPases in myocyte associated with ischemia and injury
1) Confidence 0.01 Published 2006 Journal J. Cardiovasc. Electrophysiol. Section Abstract Doc Link 16686681 Disease Relevance 0.45 Pain Relevance 0.18

General Comments

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