INT13600

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.36
First Reported 1980
Last Reported 2001
Negated 0
Speculated 0
Reported most in Abstract
Documents 4
Total Number 5
Disease Relevance 2.32
Pain Relevance 2.79

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nuclear envelope (Nrm) nucleus (Nrm)
Anatomy Link Frequency
MPO 2
neurons 1
Nrm (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Raphe magnus 55 100.00 Very High Very High Very High
Glutamate 1 99.84 Very High Very High Very High
antagonist 5 98.16 Very High Very High Very High
Central grey 16 97.86 Very High Very High Very High
Pain 6 88.64 High High
hypoalgesia 6 80.08 Quite High
tail-flick 1 77.20 Quite High
Neurotransmitter 1 75.00 Quite High
excitatory amino acid 1 74.16 Quite High
medulla 2 56.60 Quite High
Disease Link Frequency Relevance Heat
Urological Neuroanatomy 40 100.00 Very High Very High Very High
Nociception 2 94.72 High High
Pain 3 88.64 High High
Hypoalagesia 6 80.08 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In addition, since many NCF neurons are glutamatergic, we have examined the possible role of glutamate in the interaction between NCF and NRM.
NRM Binding (interaction) of in neurons associated with raphe magnus and glutamate
1) Confidence 0.36 Published 1991 Journal Brain Res. Section Abstract Doc Link 1674895 Disease Relevance 0.15 Pain Relevance 0.72
It is concluded that: (1) in response to chemical stimulation of MPO, the number of I-cells that were inhibited was more than three times the number of I-cells that were excited; in contrast, the number of E-cells that were excited was more than twice the number of E-cells that were inhibited. (2) The interaction between MPO and NRM can be modulated by blockade of the neuronal transmission or blockade of the glutamatergic system in the PAG. (3) Simultaneous activity of many synapses is required for activation of the MPO-NRM pathway. (4) MPO to NRM interaction is mediated by fibers with a conduction velocity of less than 1m/s.
NRM Binding (interaction) of in MPO associated with raphe magnus, urological neuroanatomy and central grey
2) Confidence 0.35 Published 2001 Journal Pain Section Abstract Doc Link 11690727 Disease Relevance 0.92 Pain Relevance 0.45
It is concluded that: (1) in response to chemical stimulation of MPO, the number of I-cells that were inhibited was more than three times the number of I-cells that were excited; in contrast, the number of E-cells that were excited was more than twice the number of E-cells that were inhibited. (2) The interaction between MPO and NRM can be modulated by blockade of the neuronal transmission or blockade of the glutamatergic system in the PAG. (3) Simultaneous activity of many synapses is required for activation of the MPO-NRM pathway. (4) MPO to NRM interaction is mediated by fibers with a conduction velocity of less than 1m/s.
NRM Binding (interaction) of in MPO associated with raphe magnus, urological neuroanatomy and central grey
3) Confidence 0.35 Published 2001 Journal Pain Section Abstract Doc Link 11690727 Disease Relevance 0.88 Pain Relevance 0.44
The distribution of NA terminals associated with the NRM was visualized histochemically using a glyoxylic acid-induced fluorescence technique.
NRM Binding (associated) of associated with raphe magnus
4) Confidence 0.31 Published 1980 Journal Pain Section Abstract Doc Link 7422339 Disease Relevance 0.31 Pain Relevance 0.68
The involvement of ACh in the interaction between NCF and NRM was examined with iontophoretic application of ACh and its antagonists.
NRM Binding (interaction) of associated with raphe magnus and antagonist
5) Confidence 0.12 Published 1986 Journal Brain Res. Section Abstract Doc Link 3697735 Disease Relevance 0.05 Pain Relevance 0.51

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox