INT136874

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Context Info
Confidence 0.38
First Reported 2005
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 0.19
Pain Relevance 0.05

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

lipid binding (Pebp1) mitochondrion (Pebp1) extracellular space (Pebp1)
Golgi apparatus (Pebp1) cytoplasm (Pebp1)
Anatomy Link Frequency
liver 1
Pebp1 (Mus musculus)
Pain Link Frequency Relevance Heat
Paracetamol 2 50.84 Quite High
agonist 9 5.00 Very Low Very Low Very Low
cINOD 4 5.00 Very Low Very Low Very Low
Inflammation 3 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
metalloproteinase 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Placental Insufficiency 1 95.52 Very High Very High Very High
Embryonic Lethality 1 94.28 High High
Necrosis 1 43.52 Quite Low
Ovarian Cancer 9 5.00 Very Low Very Low Very Low
Apoptosis 7 5.00 Very Low Very Low Very Low
INFLAMMATION 6 5.00 Very Low Very Low Very Low
Obesity 5 5.00 Very Low Very Low Very Low
Cancer 4 5.00 Very Low Very Low Very Low
Polycystic Ovary Syndrome 3 5.00 Very Low Very Low Very Low
Targeted Disruption 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Peroxisome proliferator-activated receptor (PPAR)-binding protein (PBP) but not PPAR-interacting protein (PRIP) is required for nuclear translocation of constitutive androstane receptor in mouse liver.
Positive_regulation (required) of PBP in liver
1) Confidence 0.38 Published 2006 Journal Biochem. Biophys. Res. Commun. Section Title Doc Link 16828057 Disease Relevance 0 Pain Relevance 0.05
These findings are consistent with the hypothesis that PBP is a required factor for PPAR?
Positive_regulation (required) of PBP
2) Confidence 0.04 Published 2005 Journal Reprod Biol Endocrinol Section Body Doc Link PMC1266036 Disease Relevance 0.19 Pain Relevance 0

General Comments

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