INT138332

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Context Info
Confidence 0.35
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.25
Pain Relevance 0.59

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (P2rx3) plasma membrane (F2rl1) signal transducer activity (F2rl1)
P2rx3 (Rattus norvegicus)
F2rl1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
agonist 4 98.40 Very High Very High Very High
IPN 1 98.32 Very High Very High Very High
Dorsal horn neuron 1 92.56 High High
adenocard 2 84.44 Quite High
nociceptor 1 76.32 Quite High
Pain 3 75.00 Quite High
Inflammation 1 73.84 Quite High
Disease Link Frequency Relevance Heat
Inflammatory Pain 1 98.32 Very High Very High Very High
Nociception 1 74.32 Quite High
INFLAMMATION 1 73.84 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These findings indicate that PAR2 agonists may potentiate the sensitivity of P2X3 ion channel to noxious stimuli, and the interaction between PAR2 and P2X3 may be an important mechanism underlying inflammatory pain.
P2X3 Binding (interaction) of PAR2 associated with ipn and agonist
1) Confidence 0.35 Published 2006 Journal Neuroreport Section Abstract Doc Link 16951565 Disease Relevance 0.25 Pain Relevance 0.59

General Comments

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