INT140531

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Context Info
Confidence 0.72
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 1.83
Pain Relevance 1.34

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Ngf) extracellular space (Ngf) aging (Ngf)
extracellular region (Ngf) molecular_function (Ngf)
Anatomy Link Frequency
nerve 1
Ngf (Rattus norvegicus)
DYT10 (Homo sapiens)
Pain Link Frequency Relevance Heat
Hyperalgesia 9 100.00 Very High Very High Very High
Kinase C 5 100.00 Very High Very High Very High
Nerve growth factor 3 100.00 Very High Very High Very High
Inflammation 1 98.84 Very High Very High Very High
Neuropathic pain 1 98.16 Very High Very High Very High
intrathecal 1 82.00 Quite High
Pain 3 81.80 Quite High
Analgesic 1 46.96 Quite Low
Disease Link Frequency Relevance Heat
Hyperalgesia 9 100.00 Very High Very High Very High
Burns 9 100.00 Very High Very High Very High
INFLAMMATION 1 98.84 Very High Very High Very High
Neuropathic Pain 1 98.16 Very High Very High Very High
Injury 5 88.72 High High
Pain 3 81.80 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Because nerve growth factor (NGF), which is elevated in burn-injured tissue, produces mechanical hyperalgesia and activates protein kinase C (PKC)-epsilon, a key mediator in inflammatory and neuropathic pain, we used this model to evaluate the role of the NGF receptor, tyrosine-receptor kinase A (TrkA), and PKC-epsilon in burn-induced primary mechanical hyperalgesia.
NGF Positive_regulation (activates) of PKC in nerve associated with kinase c, hyperalgesia, inflammation, nerve growth factor, burns and neuropathic pain
1) Confidence 0.72 Published 2006 Journal J Pain Section Abstract Doc Link 17157774 Disease Relevance 1.83 Pain Relevance 1.34

General Comments

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