INT141189

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Context Info
Confidence 0.41
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 1.42
Pain Relevance 1.14

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Bax) endoplasmic reticulum (Bax) intracellular (Bax)
cytoplasm (Bax) cytosol (Bax) cell proliferation (Bax)
Bax (Mus musculus)
Pain Link Frequency Relevance Heat
Eae 6 99.88 Very High Very High Very High
allodynia 1 99.38 Very High Very High Very High
Thermal hyperalgesia 1 98.88 Very High Very High Very High
Spinal cord 5 89.36 High High
Sciatic nerve 3 87.76 High High
Dorsal horn 2 84.96 Quite High
Neuropathic pain 4 79.56 Quite High
Pain 3 25.00 Low Low
Disease Link Frequency Relevance Heat
Injury 6 99.88 Very High Very High Very High
Neuropathic Pain 5 99.38 Very High Very High Very High
Hyperalgesia 1 98.88 Very High Very High Very High
Apoptosis 7 96.64 Very High Very High Very High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Administration of phenyl-N-tert-butylnitrone (PBN), a potent ROS scavenger, reduced the development of thermal hyperalgesia and mechanical allodynia at 1 and 3 days post-CCI, and decreased the mRNA levels of bax, apaf-1, and caspase-9.
Negative_regulation (decreased) of Transcription (levels) of bax associated with allodynia, eae and thermal hyperalgesia
1) Confidence 0.41 Published 2007 Journal Pharmacol. Res. Section Abstract Doc Link 17207636 Disease Relevance 1.42 Pain Relevance 1.14

General Comments

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