INT141256

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Context Info
Confidence 0.50
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.99
Pain Relevance 0.06

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (S100A8, VCL) extracellular region (S100A8, VCL) cytoskeleton (S100A8, VCL)
plasma membrane (S100A8, VCL) protein complex (VCL) cytosol (VCL)
Anatomy Link Frequency
gastric epithelium 1
S100A8 (Homo sapiens)
VCL (Homo sapiens)
Pain Link Frequency Relevance Heat
peptic ulcer disease 1 64.96 Quite High
Disease Link Frequency Relevance Heat
Injury 1 98.84 Very High Very High Very High
Adhesions 4 97.80 Very High Very High Very High
Disease 3 94.88 High High
Ulcers 3 75.00 Quite High
Alagille Syndrome 1 74.72 Quite High
Stomach Cancer 1 65.84 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We hypothesize that CagA-mediated inactivation of vinculin is a key step in the mechanism by which H. pylori induces damage to the gastric epithelium and represents an important step in disease development.
CagA Positive_regulation (inactivation) of vinculin in gastric epithelium associated with disease
1) Confidence 0.50 Published 2007 Journal Cell. Microbiol. Section Abstract Doc Link 17217431 Disease Relevance 0.99 Pain Relevance 0.06

General Comments

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