INT141866

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Context Info
Confidence 0.42
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.24
Pain Relevance 0.18

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Nos3) aging (Nos3) Golgi apparatus (Nos3)
cytoplasm (Nos3) signal transduction (Nos3) oxidoreductase activity (Nos3)
Anatomy Link Frequency
heart 2
Nos3 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
ischemia 6 97.04 Very High Very High Very High
IPN 1 65.84 Quite High
Disease Link Frequency Relevance Heat
Cv Unclassified Under Development 5 97.04 Very High Very High Very High
Coronary Artery Disease 1 40.44 Quite Low
Injury 1 38.76 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In conclusion, we demonstrated a novel redox mechanism in IP-induced eNOS and GLUT-4 translocation and the role of caveolar paradox in making the heart euglycemic during the process of ischemia, leading to myocardial protection in a clinically relevant rat ischemic model.
Regulation (role) of Localization (translocation) of eNOS in heart associated with ischemia
1) Confidence 0.42 Published 2007 Journal Am. J. Physiol. Heart Circ. Physiol. Section Abstract Doc Link 17277024 Disease Relevance 0.24 Pain Relevance 0.18

General Comments

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