INT143941

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.00
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 1.03
Pain Relevance 1.11

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

aging (Fos) DNA binding (Fos) response to stress (Fos)
extracellular space (Il1f5) extracellular region (Il1f5) nucleolus (Fos)
Anatomy Link Frequency
spinal 2
Il1f5 (Mus musculus)
Fos (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Inflammation 2 100.00 Very High Very High Very High
Hyperalgesia 8 99.80 Very High Very High Very High
withdrawal 2 97.40 Very High Very High Very High
Pain 2 97.04 Very High Very High Very High
antagonist 2 85.84 High High
Disease Link Frequency Relevance Heat
INFLAMMATION 2 100.00 Very High Very High Very High
Hyperalgesia 8 99.80 Very High Very High Very High
Pain 2 97.04 Very High Very High Very High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Interleukin-1ra inhibits Fos expression and hyperalgesia in rats.
Interleukin-1ra Negative_regulation (inhibits) of Gene_expression (expression) of Fos associated with hyperalgesia
1) Confidence 0.00 Published 2007 Journal Neuroreport Section Title Doc Link 17496810 Disease Relevance 0.50 Pain Relevance 0.54
The findings were that interleukin-1ra inhibited inflammation-induced Fos expression and hyperalgesia, which suggests that endogenous interleukin-1beta facilitates transmission of noxious messages at the spinal level by processes involving an enhanced Fos expression.
interleukin-1ra Negative_regulation (inhibited) of Gene_expression (expression) of Fos in spinal associated with hyperalgesia and inflammation
2) Confidence 0.00 Published 2007 Journal Neuroreport Section Abstract Doc Link 17496810 Disease Relevance 0.53 Pain Relevance 0.57

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox