INT144854

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Context Info
Confidence 0.13
First Reported 2005
Last Reported 2010
Negated 0
Speculated 1
Reported most in Body
Documents 10
Total Number 11
Disease Relevance 2.03
Pain Relevance 0.31

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (STUB1) protein binding, bridging (STUB1) enzyme binding (STUB1)
protein maturation (STUB1) ligase activity (STUB1) cytoplasm (STUB1)
Anatomy Link Frequency
HeLa 1
STUB1 (Homo sapiens)
Pain Link Frequency Relevance Heat
agonist 3 87.12 High High
Clonidine 2 86.28 High High
cytokine 24 72.48 Quite High
metalloproteinase 31 60.16 Quite High
Calcium channel 11 23.76 Low Low
aspirin 21 12.76 Low Low
Inflammation 18 5.00 Very Low Very Low Very Low
antagonist 14 5.00 Very Low Very Low Very Low
rheumatoid arthritis 10 5.00 Very Low Very Low Very Low
Osteoarthritis 10 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Prostate Cancer 83 99.84 Very High Very High Very High
Rheumatoid Arthritis 30 94.48 High High
Apoptosis 75 93.48 High High
Cancer 101 91.64 High High
Liver Cancer 1 84.08 Quite High
Acquired Immune Deficiency Syndrome Or Hiv Infection 2 76.84 Quite High
Breast Cancer 5 75.60 Quite High
Fever 4 75.00 Quite High
Colon Cancer 52 74.80 Quite High
Reprotox - General 1 31 74.24 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
2 promoter was also determined by ChIP assay in these cells.
Spec (determined) Gene_expression (assay) of ChIP
1) Confidence 0.13 Published 2005 Journal Nucleic Acids Research Section Body Doc Link PMC1084319 Disease Relevance 0.31 Pain Relevance 0
In the present study, we included analysis of the ChIP Display-disclosed genes for their expression in PCa biopsies.
Gene_expression (expression) of ChIP associated with prostate cancer
2) Confidence 0.10 Published 2007 Journal Mol Cancer Section Body Doc Link PMC1904239 Disease Relevance 0.37 Pain Relevance 0
All target genes selected for ChIP-qPCR assays were identified by microarray analysis as down-regulated in response to Par-4 overexpression and represented a spectrum of gene ontologies, including apoptosis, microRNA pathway/regulation and cell cycle (Figure 2A, Additional file 2).
Gene_expression (selected) of ChIP associated with apoptosis
3) Confidence 0.09 Published 2010 Journal Mol Cancer Section Body Doc Link PMC2883962 Disease Relevance 0.78 Pain Relevance 0
Recently, c-Jun and AP-1 have been demonstrated by genome wide ChIP-on-chip analysis to interact with ?
Gene_expression (analysis) of chip
4) Confidence 0.08 Published 2010 Journal Mol Cancer Section Body Doc Link PMC2955614 Disease Relevance 0.06 Pain Relevance 0
Further investigation was performed by using EMSA and ChIP assay, confirming that the ?
Gene_expression (assay) of ChIP
5) Confidence 0.08 Published 2010 Journal Mol Cancer Section Body Doc Link PMC2955614 Disease Relevance 0 Pain Relevance 0.03
Recently, c-Jun and AP-1 have been demonstrated by genome wide ChIP-on-chip analysis to interact with ?
Gene_expression (analysis) of ChIP
6) Confidence 0.08 Published 2010 Journal Mol Cancer Section Body Doc Link PMC2955614 Disease Relevance 0.06 Pain Relevance 0
We assayed REST occupancy at the four functional CACNA1A RE1s in HeLa by ChIP assay.
Gene_expression (assay) of ChIP in HeLa
7) Confidence 0.08 Published 2006 Journal Nucleic Acids Research Section Body Doc Link PMC1557810 Disease Relevance 0 Pain Relevance 0
On the contrary, ChIP analysis using either an HA-PPAR?
Gene_expression (analysis) of ChIP
8) Confidence 0.08 Published 2008 Journal Arthritis Res Ther Section Body Doc Link PMC2656243 Disease Relevance 0 Pain Relevance 0
Since reporter constructs driven by elements of the human MMP-1 and MMP-13 promoters often do not mirror expression of the endogenous genes, we shifted to a more direct, in vivo approach, using ChIP assays to detect changes in PPAR?
Gene_expression (assays) of ChIP
9) Confidence 0.08 Published 2008 Journal Arthritis Res Ther Section Body Doc Link PMC2656243 Disease Relevance 0 Pain Relevance 0
Microarray analysis was performed at the functional Genomics Laboratory of the University of Illinois at Urbana, Champaign, using the human genome U133 plus 2.0 chip (Affymetrix, Santa Clara, CA).
Gene_expression (using) of chip
10) Confidence 0.07 Published 2010 Journal Clinical and Developmental Immunology Section Body Doc Link PMC3022181 Disease Relevance 0.26 Pain Relevance 0.07
SIN-1 and carboxy-PTIO reduced and enhanced, respectively, the rises in NE, PGE(2), and T(c) produced by intravenous LPS.
Gene_expression (reduced) of carboxy-PTIO
11) Confidence 0.00 Published 2007 Journal Am. J. Physiol. Regul. Integr. Comp. Physiol. Section Abstract Doc Link 17584955 Disease Relevance 0.21 Pain Relevance 0.21

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