INT147248

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Context Info
Confidence 0.50
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.46
Pain Relevance 0.52

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

lipid binding (Faah) Golgi apparatus (Faah) endoplasmic reticulum (Faah)
cytoplasm (Faah)
Anatomy Link Frequency
central nervous system 2
Faah (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Analgesic 2 99.84 Very High Very High Very High
Inflammation 2 99.20 Very High Very High Very High
Endocannabinoid 1 98.40 Very High Very High Very High
antidepressant 2 98.00 Very High Very High Very High
Cannabinoid receptor 1 93.88 High High
agonist 1 93.04 High High
Pain 1 89.24 High High
Central nervous system 1 88.04 High High
Potency 1 25.00 Low Low
Disease Link Frequency Relevance Heat
INFLAMMATION 2 99.20 Very High Very High Very High
Anxiety Disorder 2 98.64 Very High Very High Very High
Pain 1 89.24 High High
Central Nervous System Disease 1 88.04 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Genetic or pharmacological inactivation of FAAH leads to analgesic, anti-inflammatory, anxiolytic, and antidepressant phenotypes in rodents without showing the undesirable side effects observed with direct cannabinoid receptor agonists, indicating that FAAH may represent an attractive therapeutic target for treatment of pain, inflammation, and other central nervous system disorders.
Positive_regulation (leads) of Positive_regulation (inactivation) of FAAH in central nervous system associated with pain, analgesic, anxiety disorder, antidepressant, inflammation, cannabinoid receptor, agonist and central nervous system
1) Confidence 0.50 Published 2007 Journal Biochemistry Section Abstract Doc Link 17949010 Disease Relevance 0.46 Pain Relevance 0.52

General Comments

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