INT149105

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Context Info
Confidence 0.43
First Reported 2008
Last Reported 2011
Negated 0
Speculated 0
Reported most in Body
Documents 3
Total Number 3
Disease Relevance 0.74
Pain Relevance 0.05

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (SOD3) mitochondrion (SOD3) extracellular space (SOD3)
oxidoreductase activity (SOD3) extracellular region (SOD3) nucleus (SOD3)
Anatomy Link Frequency
Joint 2
blood vessels 1
SOD3 (Homo sapiens)
Pain Link Frequency Relevance Heat
Bioavailability 1 99.54 Very High Very High Very High
cytokine 2 41.92 Quite Low
Pain 2 10.00 Low Low
Osteoarthritis 8 5.00 Very Low Very Low Very Low
Inflammation 1 5.00 Very Low Very Low Very Low
Angina 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Urological Neuroanatomy 6 84.76 Quite High
Stress 24 63.52 Quite High
Osteoarthritis 10 63.20 Quite High
Cardiovascular Disease 36 61.76 Quite High
Chronic Renal Failure 54 61.20 Quite High
Injury 2 50.00 Quite Low
Hypertension 7 48.48 Quite Low
Diabetes Mellitus 4 48.16 Quite Low
Hyperglycemia 1 42.80 Quite Low
Disease 4 8.56 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Joint fluid from OA subjects is characterized by significantly decreased EC-SOD levels and significant decreases in GSH, and ascorbate compared to the reference group of knee joints with pain or subacute injury but macroscopically intact cartilage.
Negative_regulation (decreases) of EC-SOD in Joint
1) Confidence 0.43 Published 2008 Journal Osteoarthr. Cartil. Section Body Doc Link 18203633 Disease Relevance 0.06 Pain Relevance 0
Joint fluid from OA subjects is characterized by significantly decreased EC-SOD levels and significant decreases in GSH, and ascorbate compared to the reference group of knee joints with pain or subacute injury but macroscopically intact cartilage.
Negative_regulation (decreased) of EC-SOD in Joint
2) Confidence 0.43 Published 2008 Journal Osteoarthr. Cartil. Section Body Doc Link 18203633 Disease Relevance 0.06 Pain Relevance 0
It has been suggested that EC-SOD is the major determinant of nitric oxide bioavailability in blood vessels, and loss of EC-SOD from vascular wall may contribute to endothelial dysfunction [73].
Negative_regulation (loss) of EC-SOD in blood vessels associated with bioavailability
3) Confidence 0.38 Published 2011 Journal Cardiology Research and Practice Section Body Doc Link PMC3022166 Disease Relevance 0.62 Pain Relevance 0.05

General Comments

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