INT149905

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Context Info
Confidence 0.43
First Reported 2008
Last Reported 2009
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0.98
Pain Relevance 0.82

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Cav3) Golgi apparatus (Cav3) plasma membrane organization (Cav3)
plasma membrane (Cav3) enzyme binding (Cav3) protein complex (Cav3)
Anatomy Link Frequency
B cells 1
Cav3 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Snapping jaw 4 94.20 High High
Hyperalgesia 5 91.48 High High
Spinal cord 1 81.20 Quite High
intrathecal 1 76.76 Quite High
Calcium channel 1 75.00 Quite High
Disease Link Frequency Relevance Heat
Temporomandibular Joint Syndrome 4 94.20 High High
Hyperalgesia 6 91.48 High High
Nociception 4 88.36 High High
Shock 1 78.72 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Observations showed immature type B cells possessed a few caveolae with Cav1 but lacked Cav3 protein at postnatal day 5 (P5).
Negative_regulation (lacked) of Cav3 protein in B cells
1) Confidence 0.43 Published 2008 Journal Anatomical record (Hoboken, N.J. : 2007) Section Abstract Doc Link 18286598 Disease Relevance 0.41 Pain Relevance 0.33
NaHS was abolished by co-administration of mibefradil, a pan-T-type Ca2+ channel inhibitor, and also suppressed by pretreatment with i.t. and i.pl. zinc chloride, known to preferentially inhibit Cav3.2 among T-type Ca2+ channel isoforms, respectively.
Negative_regulation (inhibit) of Cav3
2) Confidence 0.41 Published 2009 Journal Pain Section Abstract Doc Link 19167819 Disease Relevance 0.57 Pain Relevance 0.49

General Comments

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