INT151748

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.49
First Reported 2008
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 4
Total Number 4
Disease Relevance 4.45
Pain Relevance 5.35

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

enzyme regulator activity (PSMD2) cytosol (PSMD2) nucleoplasm (PSMD2)
small molecule metabolic process (PSMD2) cellular nitrogen compound metabolic process (PSMD2)
Anatomy Link Frequency
insular cortex 3
PPC 2
prefrontal 1
PSMD2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Anterior cingulate cortex 4 100.00 Very High Very High Very High
amygdala 2 100.00 Very High Very High Very High
Thalamus 2 100.00 Very High Very High Very High
magnetoencephalography 6 99.74 Very High Very High Very High
Pin-prick hyperalgesia 3 99.48 Very High Very High Very High
Pain 35 98.84 Very High Very High Very High
allodynia 22 98.08 Very High Very High Very High
Hyperalgesia 10 92.32 High High
c fibre 1 85.36 High High
central pain 2 80.00 Quite High
Disease Link Frequency Relevance Heat
Hyperalgesia 14 99.08 Very High Very High Very High
Pain 27 98.84 Very High Very High Very High
Neuropathic Pain 22 98.08 Very High Very High Very High
Injury 1 59.40 Quite High
Nervous System Injury 1 55.84 Quite High
INFLAMMATION 1 55.20 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Subgroup analysis using rating-weighted predictors revealed activation of the contralateral thalamus, anterior cingulate cortex, and amygdala and a bilateral activation of S1, S2, and insular cortex and prefrontal cortices in allodynia-experienced subjects.
Positive_regulation (activation) of Positive_regulation (activation) of S2 in insular cortex associated with allodynia, thalamus, amygdala and anterior cingulate cortex
1) Confidence 0.49 Published 2008 Journal J Pain Section Abstract Doc Link 18455481 Disease Relevance 1.36 Pain Relevance 1.68
We found pin-prick-induced sequential activation of contralateral S1, PPC and S2 as well as activation of ipsilateral S2 during both pin-prick hyperalgesia and normal pin-prick pain.
Positive_regulation (activation) of Positive_regulation (activation) of S2 in PPC associated with pain, hyperalgesia and pin-prick hyperalgesia
2) Confidence 0.40 Published 2010 Journal Eur J Pain Section Abstract Doc Link 19346142 Disease Relevance 1.40 Pain Relevance 1.50
MEG revealed an increased activation of bilateral secondary somatosensory cortices (S2) during condition 2 compared to condition 1.
Positive_regulation (increased) of Positive_regulation (activation) of S2 associated with magnetoencephalography
3) Confidence 0.32 Published 2009 Journal Eur J Pain Section Abstract Doc Link 19231261 Disease Relevance 0.33 Pain Relevance 0.49
Subgroup analysis using rating-weighted predictors revealed activation of the contralateral thalamus, anterior cingulate cortex, and amygdala and a bilateral activation of S1, S2, and insular cortex and prefrontal cortices in allodynia-experienced subjects.
Positive_regulation (activation) of in prefrontal Positive_regulation (activation) of S2 in insular cortex associated with allodynia, thalamus, amygdala and anterior cingulate cortex
4) Confidence 0.17 Published 2008 Journal J Pain Section Abstract Doc Link 18455481 Disease Relevance 1.36 Pain Relevance 1.68

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox