INT152276

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Context Info
Confidence 0.44
First Reported 2008
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.53
Pain Relevance 0.32

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

peptidase activity (PLG) extracellular space (PLG) extracellular region (PLG)
plasma membrane (PLG)
Anatomy Link Frequency
plasma 2
endothelial cell 2
PLG (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 11 99.60 Very High Very High Very High
bradykinin 1 94.28 High High
Pain 1 90.92 High High
rheumatoid arthritis 1 14.92 Low Low
cva 14 5.00 Very Low Very Low Very Low
Inflammatory response 3 5.00 Very Low Very Low Very Low
withdrawal 2 5.00 Very Low Very Low Very Low
corticosteroid 1 5.00 Very Low Very Low Very Low
aspirin 1 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 11 99.60 Very High Very High Very High
Increased Venous Pressure Under Development 8 97.56 Very High Very High Very High
Adhesions 8 91.96 High High
Pain 1 90.92 High High
Anti-phospholipid Antibody Syndrome 79 90.72 High High
Thrombosis 43 80.08 Quite High
Apoptosis 1 59.40 Quite High
Thrombocytopenia 4 56.40 Quite High
Cardiovascular Disease 19 50.00 Quite Low
Trepenoma Infection 1 23.20 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Plasminogen activation controls the formation of complement anaphylotoxins (responsible for vasodilatation, increase of venular permeability and leukocyte chemotaxis) and of bradykinin (which accounts for vasodilatation, increase of venular permeability and pain) by regulating the plasma contact system.
Regulation (controls) of Positive_regulation (activation) of Plasminogen in plasma associated with pain, increased venous pressure under development and bradykinin
1) Confidence 0.44 Published 2008 Journal Front. Biosci. Section Abstract Doc Link 18508538 Disease Relevance 0.64 Pain Relevance 0.32
On the other hand, the displacement of tissue type plasminogen activator (t-PA) from annexin II, an endothelial cell membrane receptor and simultaneously enhancer to t-PA [16] could reduce the plasmin activation leading in deceleration of fibrinolysis [2].
Regulation (reduce) of Positive_regulation (activation) of plasmin in endothelial cell
2) Confidence 0.09 Published 2010 Journal J Cardiothorac Surg Section Body Doc Link PMC2987921 Disease Relevance 0.89 Pain Relevance 0

General Comments

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