INT154229

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Context Info
Confidence 0.57
First Reported 2005
Last Reported 2009
Negated 0
Speculated 2
Reported most in Body
Documents 22
Total Number 25
Disease Relevance 12.50
Pain Relevance 6.19

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

lysosome (ARSD)
Anatomy Link Frequency
brain 4
anterior 1
synovial cells 1
bowel 1
ARSD (Homo sapiens)
Pain Link Frequency Relevance Heat
rheumatoid arthritis 816 99.40 Very High Very High Very High
Osteoarthritis 588 99.04 Very High Very High Very High
Inflammation 221 95.08 Very High Very High Very High
depression 4 94.24 High High
Anterior cingulate cortex 415 93.08 High High
cva 1 91.40 High High
imagery 133 89.04 High High
Leflunomide 60 87.84 High High
cINOD 72 87.28 High High
Neuropeptide 11 75.28 Quite High
Disease Link Frequency Relevance Heat
Cv General 2 Under Development 20 100.00 Very High Very High Very High
Rheumatoid Arthritis 816 99.40 Very High Very High Very High
Frailty 600 99.04 Very High Very High Very High
INFLAMMATION 221 95.08 Very High Very High Very High
Manic Depressive Disorder 4 94.60 High High
Depression 4 94.24 High High
Autism 222 94.16 High High
Measles 4 91.88 High High
Mental Disorders 1 91.88 High High
Death 1 91.76 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Direct comparisons (ASD>TD, TD>ASD) between groups revealed no significant differences for the contrast of Self>Rest in whole-brain analyses (Z>2.3, cluster significance threshold of p?
Spec (analyses) Gene_expression (comparisons) of ASD in brain
1) Confidence 0.57 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2568959 Disease Relevance 0 Pain Relevance 0
While direct statistical comparisons between groups (ASD>TD, TD>ASD) revealed no significant differences for the contrast of Other>Rest in whole-brain, cluster corrected analyses (Z>2.3, cluster significance threshold of p?
Gene_expression (groups) of ASD in brain
2) Confidence 0.57 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2568959 Disease Relevance 0 Pain Relevance 0
Direct comparisons (ASD>TD, TD>ASD) between groups revealed no significant differences for the contrast of Self>Rest in whole-brain analyses (Z>2.3, cluster significance threshold of p?
Spec (analyses) Gene_expression (comparisons) of ASD in brain
3) Confidence 0.57 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2568959 Disease Relevance 0 Pain Relevance 0
While direct statistical comparisons between groups (ASD>TD, TD>ASD) revealed no significant differences for the contrast of Other>Rest in whole-brain, cluster corrected analyses (Z>2.3, cluster significance threshold of p?
Gene_expression (groups) of ASD in brain
4) Confidence 0.57 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2568959 Disease Relevance 0 Pain Relevance 0
For a second ASD participant, data were not acquired because scanner-safe glasses of the correct prescription were not available. fMRI analyses were conducted on the remaining 10 ASD and 14 control participants.
Gene_expression (conducted) of ASD
5) Confidence 0.53 Published 2008 Journal Brain Section Body Doc Link PMC2525446 Disease Relevance 0.53 Pain Relevance 0.10
Compared with controls, ASD participants also showed significantly greater activation for correct trials in another more anterior perigenual rACC region bilaterally (Fig. 3B), and in the left hemisphere this region also showed a significantly greater response to errors (Fig. 3C).
Gene_expression (participants) of ASD in anterior
6) Confidence 0.53 Published 2008 Journal Brain Section Body Doc Link PMC2525446 Disease Relevance 0 Pain Relevance 0
In addition, the timing of the haemodynamic response to correct antisaccades in ASD, which started ?
Gene_expression (started) of ASD
7) Confidence 0.53 Published 2008 Journal Brain Section Body Doc Link PMC2525446 Disease Relevance 0.17 Pain Relevance 0.07
5, 34, 5, cluster size = 1112 mm2, CWP = 0.0001; right maximum 9, 38, 12, cluster size = 850 mm2, CWP = 0.0001), only the ASD group showed a significant response to correct trials (left maximum ?
Gene_expression (group) of ASD
8) Confidence 0.53 Published 2008 Journal Brain Section Body Doc Link PMC2525446 Disease Relevance 0 Pain Relevance 0.03
Comparisons were restricted to antisaccade trials since healthy and ASD participants produce too few prosaccade errors with this paradigm (Manoach et al., 2004).
Gene_expression (produce) of ASD
9) Confidence 0.53 Published 2008 Journal Brain Section Body Doc Link PMC2525446 Disease Relevance 0.05 Pain Relevance 0.19
Subsequent investigations found no associations between MV exposure and ASD but did not test for the presence of MV RNA in bowel or focus on children with ASD and GI disturbances.
Gene_expression (exposure) of ASD in bowel
10) Confidence 0.44 Published 2008 Journal PLoS ONE Section Abstract Doc Link PMC2526159 Disease Relevance 0.97 Pain Relevance 0.03
-dihydro-ASD produced was higher in RA as compared with OA (Fig. 3a).
Gene_expression (produced) of ASD associated with rheumatoid arthritis and osteoarthritis
11) Confidence 0.38 Published 2005 Journal Arthritis Res Ther Section Body Doc Link PMC1257423 Disease Relevance 0.48 Pain Relevance 0.34
Radiolabeled ASD was converted into 5?
Gene_expression (converted) of ASD
12) Confidence 0.38 Published 2005 Journal Arthritis Res Ther Section Body Doc Link PMC1257423 Disease Relevance 0.37 Pain Relevance 0.29
There was no significant gender difference in enzyme activity in the control group, neither was there any great variation but in the ASD group there was a significant variation, randomly distributed between the sexes.
Gene_expression (group) of ASD
13) Confidence 0.34 Published 2005 Journal BMC Psychiatry Section Body Doc Link PMC1190193 Disease Relevance 0.35 Pain Relevance 0.08
Conversion of ASD and testosterone into downstream steroid hormones in mixed synovial cells
Gene_expression (Conversion) of ASD in synovial cells
14) Confidence 0.33 Published 2005 Journal Arthritis Res Ther Section Body Doc Link PMC1257423 Disease Relevance 0.52 Pain Relevance 0.38
DHEA is the major delta 5 androgen (Fig. 1), which is converted to androstenediol, ASD, 16?
Gene_expression (DHEA) of ASD
15) Confidence 0.33 Published 2005 Journal Arthritis Res Ther Section Body Doc Link PMC1257423 Disease Relevance 0.70 Pain Relevance 0.45
Radiolabeled testosterone was converted to ASD, 5?
Gene_expression (converted) of ASD
16) Confidence 0.33 Published 2005 Journal Arthritis Res Ther Section Body Doc Link PMC1257423 Disease Relevance 0.48 Pain Relevance 0.42
DHEA is the major delta 5 androgen (Fig. 1), which is converted to androstenediol, ASD, 16?
Gene_expression (is) of ASD
17) Confidence 0.33 Published 2005 Journal Arthritis Res Ther Section Body Doc Link PMC1257423 Disease Relevance 0.70 Pain Relevance 0.45
-dihydro-ASD, testosterone, and negligible amounts of E1, E2, E3, or 16?
Gene_expression (amounts) of ASD
18) Confidence 0.33 Published 2005 Journal Arthritis Res Ther Section Abstract Doc Link PMC1257423 Disease Relevance 1.64 Pain Relevance 0.82
The third androgen, testosterone, was converted into ASD, 5?
Gene_expression (converted) of ASD
19) Confidence 0.33 Published 2005 Journal Arthritis Res Ther Section Abstract Doc Link PMC1257423 Disease Relevance 1.71 Pain Relevance 0.86
-dihydro-ASD, testosterone, and negligible amounts of E1, E2, E3, and 16?
Gene_expression (testosterone) of ASD
20) Confidence 0.33 Published 2005 Journal Arthritis Res Ther Section Body Doc Link PMC1257423 Disease Relevance 0.37 Pain Relevance 0.29

General Comments

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