INT154451

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Context Info
Confidence 0.12
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 5
Disease Relevance 0.35
Pain Relevance 0.50

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (SLC38A3) transmembrane transport (SLC38A3)
Anatomy Link Frequency
brain 1
SLC38A3 (Homo sapiens)
Pain Link Frequency Relevance Heat
Opioid 6 98.80 Very High Very High Very High
tolerance 12 30.60 Quite Low
Pain 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Fever 48 79.72 Quite High
Stress 20 37.20 Quite Low
Pain 4 5.00 Very Low Very Low Very Low
Sprains And Strains 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
[Author's response: Indeed, the primary divide between sn1,2 and sn2,3 lipids may not be the direct result of selection.
Gene_expression (lipids) of sn1
1) Confidence 0.12 Published 2008 Journal Biol Direct Section Body Doc Link PMC2478661 Disease Relevance 0.14 Pain Relevance 0
The occurrence of the divide between sn1,2 (ester/ether-fatty acid) and sn2,3 ether-isoprenoid lipids
Gene_expression (ether) of sn1
2) Confidence 0.12 Published 2008 Journal Biol Direct Section Body Doc Link PMC2478661 Disease Relevance 0.13 Pain Relevance 0
Of course, in this view, the so-called "primary divide" between sn1,2 and sn2,3 glycerol lipids now appears as secondary!
Gene_expression (lipids) of sn1
3) Confidence 0.12 Published 2008 Journal Biol Direct Section Body Doc Link PMC2478661 Disease Relevance 0 Pain Relevance 0
In the meantime some sn1,2 glycerol isoprenoid ether lipids could have been produced, to disappear later on with the recruitment of G1PDH as a enzyme providing a more adequate substrate.
Gene_expression (produced) of sn1
4) Confidence 0.12 Published 2008 Journal Biol Direct Section Body Doc Link PMC2478661 Disease Relevance 0.08 Pain Relevance 0
While L-kyotorphin is a stimulator of opioid peptide transport, it is a transportable substrate for the H+-coupled peptide transporter PEPT2, which is expressed widely in the brain.
Gene_expression (expressed) of H+-coupled in brain associated with opioid
5) Confidence 0.05 Published 2008 Journal Drug Metab. Pharmacokinet. Section Abstract Doc Link 18762712 Disease Relevance 0 Pain Relevance 0.50

General Comments

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