INT154586

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Context Info
Confidence 0.28
First Reported 2008
Last Reported 2008
Negated 1
Speculated 0
Reported most in Abstract
Documents 1
Total Number 3
Disease Relevance 0.22
Pain Relevance 0.59

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transmembrane transport (Kcna3)
Anatomy Link Frequency
neuronal 2
interneurons 2
Kcna3 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
GABAergic 3 98.08 Very High Very High Very High
Glutamate receptor 3 97.92 Very High Very High Very High
tetrodotoxin 3 90.64 High High
potassium channel 3 88.84 High High
Action potential 3 84.20 Quite High
Disease Link Frequency Relevance Heat
Targeted Disruption 3 84.36 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The results show that Kv3.1b/3.2 expression is differentially controlled by neuronal activity and neurotrophic factors.
Regulation (controlled) of Gene_expression (expression) of Kv3 in neuronal
1) Confidence 0.28 Published 2008 Journal Neuroscience Section Abstract Doc Link 18775767 Disease Relevance 0.05 Pain Relevance 0.11
However, chronic activity deprivation failed to alter Kv3.1b and marginally delayed Kv3.2 protein expression.
Neg (failed) Regulation (alter) of Gene_expression (expression) of Kv3
2) Confidence 0.12 Published 2008 Journal Neuroscience Section Abstract Doc Link 18775767 Disease Relevance 0.08 Pain Relevance 0.25
Neuronal activity and TrkB ligands influence Kv3.1b and Kv3.2 expression in developing cortical interneurons.
Regulation (influence) of Gene_expression (expression) of Kv3 in interneurons
3) Confidence 0.12 Published 2008 Journal Neuroscience Section Title Doc Link 18775767 Disease Relevance 0.08 Pain Relevance 0.22

General Comments

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