INT156239

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Context Info
Confidence 0.09
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 2.40
Pain Relevance 0.81

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Tnfsf14, F2rl1) signal transduction (F2rl1) extracellular space (Tnfsf14)
extracellular region (Tnfsf14) signal transducer activity (F2rl1)
Anatomy Link Frequency
endothelial cells 1
monocytes 1
Tnfsf14 (Mus musculus)
F2rl1 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 16 98.84 Very High Very High Very High
chemokine 2 92.20 High High
Angina 2 85.44 High High
cytokine 2 69.28 Quite High
Disease Link Frequency Relevance Heat
INFLAMMATION 14 98.84 Very High Very High Very High
Cancer 4 96.60 Very High Very High Very High
Necrosis 4 96.36 Very High Very High Very High
Cv General 3 Under Development 2 85.44 High High
Vasculitis 2 80.60 Quite High
Atherosclerosis 10 78.48 Quite High
Atherosclerotic Plaque 2 63.44 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
A LIGHT-mediated upregulation of PAR-2 mRNA levels was also found in human monocytes when these cells were preactivated by tumor necrosis factor alpha.
LIGHT Positive_regulation (upregulation) of PAR-2 in monocytes associated with necrosis and cancer
1) Confidence 0.09 Published 2009 Journal Circ. Res. Section Abstract Doc Link 19023130 Disease Relevance 1.25 Pain Relevance 0.41
We showed that the LIGHT-mediated upregulation of PAR-2 in endothelial cells is mediated through the HVEM receptor, involving Jun N-terminal kinase signaling pathways.
LIGHT Positive_regulation (upregulation) of PAR-2 in endothelial cells
2) Confidence 0.09 Published 2009 Journal Circ. Res. Section Abstract Doc Link 19023130 Disease Relevance 1.14 Pain Relevance 0.39

General Comments

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